| Cardiac structure and function in renal disease. | |
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MedLine Citation:
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PMID: 8834168 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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Cardiac death secondary to ischaemia, but not necessarily associated with coronary lesions, is the leading cause of death in uraemic patients. This article reviews recent findings that demonstrate abnormalities of cardiac structure which have a bearing on ischaemia tolerance. These abnormalities comprise the following: (1) left ventricular hypertrophy not fully explained by increased mean arterial pressure, (2) interstitial fibrosis and (3) abnormalities of myocardial microvasculature, i.e. capillary rarefaction and arteriolar-wall thickening. |
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Authors:
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K Amann; E Ritz |
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Publication Detail:
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Type: Journal Article; Research Support, Non-U.S. Gov't; Review |
Journal Detail:
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Title: Current opinion in nephrology and hypertension Volume: 5 ISSN: 1062-4821 ISO Abbreviation: Curr. Opin. Nephrol. Hypertens. Publication Date: 1996 Jan |
Date Detail:
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Created Date: 1996-12-03 Completed Date: 1996-12-03 Revised Date: 2006-11-15 |
Medline Journal Info:
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Nlm Unique ID: 9303753 Medline TA: Curr Opin Nephrol Hypertens Country: UNITED STATES |
Other Details:
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Languages: eng Pagination: 102-6 Citation Subset: IM |
Affiliation:
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Department of Pathology, University of Heidelberg, Germany. |
Export Citation:
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APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
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Endomyocardial Fibrosis
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etiology,
pathology,
physiopathology* Humans Hypertrophy, Left Ventricular / etiology, pathology, physiopathology* Risk Factors Uremia / complications, pathology, physiopathology* |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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