Document Detail


Captopril and the maintenance of blood pressure after sinoaortic denervation in the rabbit.
MedLine Citation:
PMID:  6269679     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
1. At a dose of 1 mg/kg intravenously, captopril lowered blood pressure in conscious rabbits, that had undergone denervation of sinoaortic baroreceptors, but had no effect on heart rate. In sham-operated controls, this dose caused only an increase in heart rate. 2. In the same experiments, captopril caused a substantial inhibition of plasma angiotensin converting enzyme (CE) activity of rapid onset and gradual decline over 2 to 3 h. The time course of recovery of blood pressure and plasma CE activity were similar. 3. Saralasin, by intravenous infusion, lowered blood pressure in sinoaortic rabbits but to a smaller extent than captopril. 4. The fall in blood pressure observed after captopril is not dependent on the integrity of baroreceptor afferents.
Authors:
M A Petty; J L Reid
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  British journal of pharmacology     Volume:  72     ISSN:  0007-1188     ISO Abbreviation:  Br. J. Pharmacol.     Publication Date:  1981 Apr 
Date Detail:
Created Date:  1981-12-15     Completed Date:  1981-12-15     Revised Date:  2009-11-18    
Medline Journal Info:
Nlm Unique ID:  7502536     Medline TA:  Br J Pharmacol     Country:  ENGLAND    
Other Details:
Languages:  eng     Pagination:  645-7     Citation Subset:  IM    
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MeSH Terms
Descriptor/Qualifier:
Angiotensin-Converting Enzyme Inhibitors
Animals
Aorta, Thoracic / innervation*
Blood Pressure / drug effects*
Captopril / pharmacology*
Carotid Sinus / innervation*
Denervation
Heart Rate / drug effects
Male
Pressoreceptors / physiology
Proline / analogs & derivatives*
Rabbits
Saralasin / pharmacology
Chemical
Reg. No./Substance:
0/Angiotensin-Converting Enzyme Inhibitors; 147-85-3/Proline; 34273-10-4/Saralasin; 62571-86-2/Captopril
Comments/Corrections

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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