Document Detail


Candida species differ in their interactions with immature human gastrointestinal epithelial cells.
MedLine Citation:
PMID:  21283049     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Life-threatening gastrointestinal (GI) diseases of prematurity are highly associated with systemic candidiasis. This implicates the premature GI tract as an important site for invasion by Candida. Invasive interactions of Candida spp. with immature enterocytes have heretofore not been analyzed. Using a primary immature human enterocyte line, we compared the ability of multiple isolates of different Candida spp. to penetrate, injure, and induce a cytokine response from host cells. Of all the Candida spp. analyzed, C. albicans had the greatest ability to penetrate and injure immature enterocytes and to elicit IL-8 release (p < 0.01). In addition, C. albicans was the only Candida spp. to form filamentous hyphae when in contact with immature enterocytes. Similarly, a C. albicans mutant with defective hyphal morphogenesis and invasiveness had attenuated cytotoxicity for immature enterocytes (p < 0.003). Thus, hyphal morphogenesis correlates with immature enterocyte penetration, injury, and inflammatory responses. Furthermore, variability in enterocyte injury was observed among hyphal-producing C. albicans strains, suggesting that individual organism genotypes also influence host-pathogen interactions. Overall, the finding that Candida spp. differed in their interactions with immature enterocytes implicates that individual spp. may use different pathogenesis mechanisms.
Authors:
Christina Falgier; Sara Kegley; Heather Podgorski; Timothy Heisel; Kathleen Storey; Catherine M Bendel; Cheryl A Gale
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Publication Detail:
Type:  Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Pediatric research     Volume:  69     ISSN:  1530-0447     ISO Abbreviation:  Pediatr. Res.     Publication Date:  2011 May 
Date Detail:
Created Date:  2011-04-15     Completed Date:  2011-07-25     Revised Date:  2012-05-02    
Medline Journal Info:
Nlm Unique ID:  0100714     Medline TA:  Pediatr Res     Country:  United States    
Other Details:
Languages:  eng     Pagination:  384-9     Citation Subset:  IM    
Affiliation:
Department of Pediatrics, University of Minnesota Medical School, Minneapolis, Minnesota, USA.
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MeSH Terms
Descriptor/Qualifier:
Bacterial Adhesion
Candida / classification,  genetics,  pathogenicity*
Cell Line
Enterocytes / immunology,  microbiology*,  pathology
Genotype
Host-Pathogen Interactions* / genetics
Humans
Hyphae
Inflammation Mediators / metabolism
Interleukin-8 / metabolism
Mutation
Phenotype
Grant Support
ID/Acronym/Agency:
AI057440/AI/NIAID NIH HHS; R01 AI057440-04/AI/NIAID NIH HHS
Chemical
Reg. No./Substance:
0/IL8 protein, human; 0/Inflammation Mediators; 0/Interleukin-8

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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