Document Detail


Calculated effect of fluid retention upon velocity of blood flow and turbulence: implications for atherosclerosis.
MedLine Citation:
PMID:  21339628     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Fluid retention increases intravascular volume and pressure. The calculations in this paper demonstrate that fluid retention increases the likelihood that blood will flow in a turbulent manner, in part due to an increase in stroke volume, regardless of whether or not blood pressure is increased. Increased turbulence will promote endothelial dysfunction, thereby contributing to the development of atherosclerotic cardiovascular disease. Accordingly, fluid retention is predictably detrimental to the cardiovascular system. The reason that some medications, such as cyclo-oxygenase-2 inhibitors, nonselective nonsteroidal anti-inflammatory drugs, estrogens, progestins, and rosiglitazone, are associated with an increased risk of myocardial infarcts and strokes may be that they cause fluid retention. Increased stroke volume and/or edema formation may indicate that a medication increases the risk of adverse cardiovascular events. For drugs that increase the risk of adverse cardiovascular events, it may be possible to reduce or neutralize the increased risk by simultaneously administering a diuretic.
Authors:
Robert P Blankfield
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Clinical hemorheology and microcirculation     Volume:  47     ISSN:  1875-8622     ISO Abbreviation:  Clin. Hemorheol. Microcirc.     Publication Date:  2011  
Date Detail:
Created Date:  2011-02-22     Completed Date:  2011-06-01     Revised Date:  2011-10-07    
Medline Journal Info:
Nlm Unique ID:  9709206     Medline TA:  Clin Hemorheol Microcirc     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  79-86     Citation Subset:  IM    
Affiliation:
Department of Family Medicine, Case Western Reserve University School of Medicine, Berea, OH, USA. blankfield@roadrunner.com
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MeSH Terms
Descriptor/Qualifier:
Anti-Inflammatory Agents, Non-Steroidal / adverse effects
Atherosclerosis / physiopathology*
Blood Flow Velocity*
Cyclooxygenase 2 Inhibitors / adverse effects
Humans
Models, Cardiovascular
Myocardial Infarction / chemically induced
Stroke / chemically induced
Chemical
Reg. No./Substance:
0/Anti-Inflammatory Agents, Non-Steroidal; 0/Cyclooxygenase 2 Inhibitors

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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