| Calcium deposition and associated chronic diseases (atherosclerosis, diffuse idiopathic skeletal hyperostosis, and others). | |
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MedLine Citation:
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PMID: 16716887 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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Extracellular matrix mineralization or calcification occurs in many pathologic conditions, including atherosclerosis, medial wall calcification, diffuse idiopathic skeletal hyperostosis, and chondrocalcinosis. Vascular wall calcification is the most common and involves two mechanisms: passive calcification resulting from breakdown of the protection system and active calcification resulting from transdifferentiation of mesenchymal cells in the vascular wall to bone. Although reports are conflicting, several matrix proteins are identified as protective factors against dystrophic calcification in nonosseous tissues. Serum matrix Gla protein may be a marker of osteometabolic syndromes that cause hyperostosis and plays a role in Milwaukee shoulder syndrome. |
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Authors:
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Fabiola Atzeni; Piercarlo Sarzi-Puttini; Maorizio Bevilacqua |
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Publication Detail:
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Type: Journal Article; Review |
Journal Detail:
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Title: Rheumatic diseases clinics of North America Volume: 32 ISSN: 0889-857X ISO Abbreviation: Rheum. Dis. Clin. North Am. Publication Date: 2006 May |
Date Detail:
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Created Date: 2006-05-23 Completed Date: 2006-07-25 Revised Date: - |
Medline Journal Info:
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Nlm Unique ID: 8708093 Medline TA: Rheum Dis Clin North Am Country: United States |
Other Details:
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Languages: eng Pagination: 413-26, viii Citation Subset: IM |
Affiliation:
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Rheumatology Unit, L Sacco University Hospital, Milan, Italy. |
Export Citation:
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| MeSH Terms | |
Descriptor/Qualifier:
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Atherosclerosis
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complications*,
metabolism,
pathology Blood Vessels / metabolism, pathology Calcinosis / complications*, metabolism, pathology Chondrocalcinosis / complications*, metabolism, pathology Humans Hyperostosis, Diffuse Idiopathic Skeletal / complications*, metabolism, pathology |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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