| Burn sera mediated effects on the E-rosetting property of normal active T1 cells. | |
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MedLine Citation:
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PMID: 3879194 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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This study investigates the suppressive effects of burn sera on normal active T1 cells. The results revealed that burn sera collected from survivors and non-survivors during the initial 'shock' phase caused varied effects on the 'E' rosetting properties of normal active T1 cells. However, unlike non-survivor sera convalescent or recovery sera failed to block the 'E' rosetting property of T1 cells. Furthermore, cold incubation in fetal calf sera (FCS) failed to abrogate the loss of T1 cells which were exposed to non-survivor (early) sera; these samples also revealed the presence of antigenic factors. Our experiments were extended to investigate the mechanisms resulting in the irreversible change, and showed that the inhibiting factors could be acting at the microtubular level of the cell cortex. |
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Authors:
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S S Mistry; N F Mistry; S Arora; N H Antia |
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Publication Detail:
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Type: Journal Article; Research Support, Non-U.S. Gov't |
Journal Detail:
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Title: Burns, including thermal injury Volume: 12 ISSN: 0305-4179 ISO Abbreviation: Burns Incl Therm Inj Publication Date: 1985 Dec |
Date Detail:
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Created Date: 1986-04-15 Completed Date: 1986-04-15 Revised Date: 2006-11-15 |
Medline Journal Info:
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Nlm Unique ID: 7512054 Medline TA: Burns Incl Therm Inj Country: ENGLAND |
Other Details:
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Languages: eng Pagination: 97-101 Citation Subset: IM |
Export Citation:
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APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
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Animals Burns / blood*, immunology, mortality Cattle Cell Migration Inhibition Colchicine / pharmacology Humans Immunity, Cellular Leukocytes / immunology Phytohemagglutinins / pharmacology Receptors, Immunologic / biosynthesis Rosette Formation* T-Lymphocytes / immunology* |
| Chemical | |
Reg. No./Substance:
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0/Phytohemagglutinins; 0/Receptors, Immunologic; 64-86-8/Colchicine |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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