Document Detail


Brain energy metabolism in angiotensin-induced acute hypertension in rats.
MedLine Citation:
PMID:  888709     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
In order to study if rapid elevation of blood pressure is associated with cerebral ischemia, anesthetized (70% N2O) and artificially ventilated rats were subjected to angiotensin-induced hypertension. After a 5 min hypertensive period, cerebral cortex tissue was frozen in situ for subsequent measurements of labile glycolytic metabolites, ammonia, and organic phosphates. The degree of hypertension induced, which gave evidence of blood-brain barrier damage in 7 of 8 rats, did not affect the tissue concentrations of labile metabolites. It is concluded that ischemia does not contribute to the barrier damage, nor is it likely to be the cause of the clinical symptoms that may occur in conscious rats in the same experimental model.
Authors:
B B Johansson; B K Siesjö
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Acta physiologica Scandinavica     Volume:  100     ISSN:  0001-6772     ISO Abbreviation:  Acta Physiol. Scand.     Publication Date:  1977 Jun 
Date Detail:
Created Date:  1977-09-29     Completed Date:  1977-09-29     Revised Date:  2003-11-14    
Medline Journal Info:
Nlm Unique ID:  0370362     Medline TA:  Acta Physiol Scand     Country:  SWEDEN    
Other Details:
Languages:  eng     Pagination:  182-6     Citation Subset:  IM    
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MeSH Terms
Descriptor/Qualifier:
Acute Disease
Ammonia / metabolism
Angiotensin II*
Animals
Blood-Brain Barrier
Brain / metabolism*
Energy Metabolism* / drug effects
Glucose / metabolism
Glycogen / metabolism
Hypertension / chemically induced,  metabolism*
Ischemic Attack, Transient / etiology,  metabolism
Male
Organophosphorus Compounds / metabolism
Rats
Rats, Inbred Strains
Chemical
Reg. No./Substance:
0/Organophosphorus Compounds; 11128-99-7/Angiotensin II; 50-99-7/Glucose; 7664-41-7/Ammonia; 9005-79-2/Glycogen

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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