Document Detail


Brain damage in preterm newborns: biological response modification as a strategy to reduce disabilities.
MedLine Citation:
PMID:  10753239     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Substances that promote the growth and maturation of oligodendrocytes and their precursors might protect against white matter injury. We suggest that neuroprotection can also be provided by such modulators of fetal and neonatal inflammatory responses as antiinflammatory cytokines, cytokine-binding proteins, and cytokine-receptor blockers. We briefly describe inflammatory responses in the fetus and newborn and show how they might contribute to brain damage. We conclude with the possibility that so-called biological response modifiers, which are drugs that modulate these inflammatory responses, might reduce the risk of brain damage and disabilities.
Authors:
O Dammann; A Leviton
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Review    
Journal Detail:
Title:  The Journal of pediatrics     Volume:  136     ISSN:  0022-3476     ISO Abbreviation:  J. Pediatr.     Publication Date:  2000 Apr 
Date Detail:
Created Date:  2000-05-03     Completed Date:  2000-05-03     Revised Date:  2007-11-15    
Medline Journal Info:
Nlm Unique ID:  0375410     Medline TA:  J Pediatr     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  433-8     Citation Subset:  AIM; IM    
Affiliation:
Neuroepidemiology Unit, Department of Neurology, Children's Hospital, Harvard Medical School, Boston, MA 02115, USA.
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MeSH Terms
Descriptor/Qualifier:
Brain Damage, Chronic / etiology,  prevention & control*
Humans
Immunologic Factors / therapeutic use*
Infant, Newborn
Infant, Premature
Infant, Premature, Diseases / drug therapy*,  etiology
Mentally Disabled Persons*
Chemical
Reg. No./Substance:
0/Immunologic Factors

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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