Document Detail


Bovine serum albumin preparations enhance in vitro production of tumor necrosis factor alpha by murine macrophages.
MedLine Citation:
PMID:  8543338     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Tumor necrosis factor alpha (TNF-alpha) is most commonly produced by macrophages stimulated by lipopolysaccharide (LPS). The present study shows that BSA in place of FBS in RPMI 1640 medium accelerated the rate of LPS-induced TNF-alpha production by resident peritoneal macrophages from BALB/c mice when compared to LPS in serum free medium. Using 10 or 100 ng LPS/ml and 100 U IFN-gamma/ml in RPMI 1640 medium plus 0.5% BSA, both cytoplasmic TNF-alpha mRNA and TNF-alpha precursor and extracellular TNF-alpha production by mouse macrophages were increased when compared to stimulation by LPS plus IFN-gamma in medium without BSA and FBS. The level of TNF-alpha produced was shown to be related to the BSA concentration. Medium containing BSA but no LPS also stimulated macrophages to produce TNF-alpha, but BSA's TNF-alpha inducing activity varied among different lots and was not blocked by polyclonal antibody to BSA. This effect appeared to be associated with the presence of immunoglobulin in BSA products. Confirmation that BSA activity was not due to LPS contamination was achieved by testing macrophages from LPS-nonresponder C3H/HeJ mice, as well as testing TNF-alpha induction in the presence of polymyxin B (10 micrograms/ml), an LPS inhibitor.
Authors:
Z M Zheng; S C Specter; G Lancz
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Publication Detail:
Type:  Comparative Study; Journal Article; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Immunological investigations     Volume:  24     ISSN:  0882-0139     ISO Abbreviation:  Immunol. Invest.     Publication Date:  1995 Aug 
Date Detail:
Created Date:  1996-02-13     Completed Date:  1996-02-13     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  8504629     Medline TA:  Immunol Invest     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  737-56     Citation Subset:  IM    
Affiliation:
Department of Medical Microbiology & Immunology, College of Medicine, University of South Florida, Tampa 33612, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Cattle
Culture Media / pharmacology*
Drug Synergism
Fatty Acids / pharmacology
Female
Gene Expression Regulation / drug effects*
Image Processing, Computer-Assisted
Immunoglobulin G / pharmacology
Interferon-gamma, Recombinant / pharmacology
Lipopolysaccharides / pharmacology*
Macrophage Activation / drug effects*
Macrophages, Peritoneal / drug effects,  metabolism*
Mice
Mice, Inbred BALB C
Mice, Inbred C3H
Polymyxin B / pharmacology
Protein Biosynthesis / drug effects
Serum Albumin, Bovine / pharmacology*
Stimulation, Chemical
Transcription, Genetic / drug effects
Tumor Necrosis Factor-alpha / biosynthesis*,  genetics
Grant Support
ID/Acronym/Agency:
DA04141/DA/NIDA NIH HHS; DA05363/DA/NIDA NIH HHS; DA05794/DA/NIDA NIH HHS
Chemical
Reg. No./Substance:
0/Culture Media; 0/Fatty Acids; 0/Immunoglobulin G; 0/Interferon-gamma, Recombinant; 0/Lipopolysaccharides; 0/Serum Albumin, Bovine; 0/Tumor Necrosis Factor-alpha; 1404-26-8/Polymyxin B

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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