Document Detail


The Bloom syndrome protein limits the lethality associated with RAD51 deficiency.
MedLine Citation:
PMID:  20215422     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Little is known about the functional interaction between the Bloom's syndrome protein (BLM) and the recombinase RAD51 within cells. Using RNA interference technology, we provide the first demonstration that RAD51 acts upstream from BLM to prevent anaphase bridge formation. RAD51 downregulation was associated with an increase in the frequency of BLM-positive anaphase bridges, but not of BLM-associated ultrafine bridges. Time-lapse live microscopy analysis of anaphase bridge cells revealed that BLM promoted cell survival in the absence of Rad51. Our results directly implicate BLM in limiting the lethality associated with RAD51 deficiency through the processing of anaphase bridges resulting from the RAD51 defect. These findings provide insight into the molecular basis of some cancers possibly associated with variants of the RAD51 gene family.
Authors:
Kenza Lahkim Bennani-Belhaj; Sébastien Rouzeau; Géraldine Buhagiar-Labarchède; Pauline Chabosseau; Rosine Onclercq-Delic; Emilie Bayart; Fabrice Cordelières; Jérôme Couturier; Mounira Amor-Guéret
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-03-09
Journal Detail:
Title:  Molecular cancer research : MCR     Volume:  8     ISSN:  1557-3125     ISO Abbreviation:  Mol. Cancer Res.     Publication Date:  2010 Mar 
Date Detail:
Created Date:  2010-03-18     Completed Date:  2010-07-12     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101150042     Medline TA:  Mol Cancer Res     Country:  United States    
Other Details:
Languages:  eng     Pagination:  385-94     Citation Subset:  IM    
Affiliation:
Institut Curie, Centre de Recherche, Centre Universitaire, Orsay, France.
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MeSH Terms
Descriptor/Qualifier:
Anaphase / genetics
Cell Death / genetics
Cell Survival / genetics
Down-Regulation / genetics
Gene Expression Regulation, Neoplastic / genetics
Hela Cells
Humans
Neoplasms / genetics*,  metabolism
RNA Interference / physiology
Rad51 Recombinase / genetics*
RecQ Helicases / genetics*,  metabolism
Sister Chromatid Exchange / genetics
Chemical
Reg. No./Substance:
EC 2.7.7.-/Rad51 Recombinase; EC 3.6.1.-/Bloom syndrome protein; EC 3.6.1.-/RecQ Helicases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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