Document Detail


Blood genomic expression profile for neuronal injury.
MedLine Citation:
PMID:  12621306     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
This study determined whether stroke and other types of insults produced a gene expression profile in blood that correlated with the presence of neuronal injury. Adult rats were subjected to ischemic stroke, intracerebral hemorrhage, status epilepticus, and insulin-induced hypoglycemia and compared with untouched, sham surgery, and hypoxia animals that had no brain injury. One day later, microarray analyses showed that 117 genes were upregulated and 80 genes were downregulated in mononuclear blood cells of the "injury" (n = 12) compared with the "no injury" (n = 9) animals. A second experiment examined the whole blood genomic response of adult rats after global ischemia and kainate seizures. Animals with no brain injury were compared with those with brain injury documented by TUNEL and PANT staining. One day later, microarray analyses showed that 37 genes were upregulated and 67 genes were downregulated in whole blood of the injury (n = 4) animals compared with the no-injury (n = 4) animals. Quantitative reverse transcription-polymerase chain reaction confirmed that the vesicular monoamine transporter-2 increased 2.3- and 1.6-fold in animals with severe and mild brain injury, respectively, compared with no-injury animals. Vascular tyrosine phosphatase-1 increased 2.0-fold after severe injury compared with no injury. The data support the hypothesis that there is a peripheral blood genomic response to neuronal injury, and that this blood response is associated with a specific blood mRNA gene expression profile that can be used as a marker of the neuronal damage.
Authors:
Yang Tang; Alex C Nee; Aigang Lu; Ruiqiong Ran; Frank R Sharp
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism     Volume:  23     ISSN:  0271-678X     ISO Abbreviation:  J. Cereb. Blood Flow Metab.     Publication Date:  2003 Mar 
Date Detail:
Created Date:  2003-03-06     Completed Date:  2003-04-08     Revised Date:  2008-11-21    
Medline Journal Info:
Nlm Unique ID:  8112566     Medline TA:  J Cereb Blood Flow Metab     Country:  United States    
Other Details:
Languages:  eng     Pagination:  310-9     Citation Subset:  IM    
Affiliation:
Department of Neurology, Neuroscience Program, University of Cincinnati, Vontz Center for Molecular Studies-Room 2327, 3125 Eden Avenue, Cincinnati, OH 45267-0536, USA. tangyy@email.uc.edu
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MeSH Terms
Descriptor/Qualifier:
Animals
Blood Physiological Phenomena
Brain Diseases / blood*,  genetics*
Down-Regulation
Gene Expression Profiling*
Genome*
Ischemic Attack, Transient / pathology,  physiopathology
Kainic Acid
Male
Membrane Glycoproteins / genetics,  metabolism
Membrane Transport Proteins*
Monocytes / physiology
Neuropeptides*
Oligonucleotide Array Sequence Analysis
Protein Phosphatase 1
Protein Tyrosine Phosphatases / genetics
RNA, Messenger / metabolism
Rats
Rats, Sprague-Dawley
Reference Values
Reverse Transcriptase Polymerase Chain Reaction
Seizures / chemically induced,  pathology,  physiopathology
Vesicular Biogenic Amine Transport Proteins
Vesicular Monoamine Transport Proteins
Grant Support
ID/Acronym/Agency:
AG 19561/AG/NIA NIH HHS; NS 28167/NS/NINDS NIH HHS; NS 38084/NS/NINDS NIH HHS; NS 38743/NS/NINDS NIH HHS
Chemical
Reg. No./Substance:
0/Membrane Glycoproteins; 0/Membrane Transport Proteins; 0/Neuropeptides; 0/RNA, Messenger; 0/Vesicular Biogenic Amine Transport Proteins; 0/Vesicular Monoamine Transport Proteins; 487-79-6/Kainic Acid; EC 3.1.3.16/Protein Phosphatase 1; EC 3.1.3.48/Protein Tyrosine Phosphatases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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