Document Detail


Blocking the deadly effects of the NMDA receptor in stroke.
MedLine Citation:
PMID:  20141829     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Excessive activation of NMDA glutamate receptors contributes to neuronal death after stroke. In this issue, Tu et al. (2010) demonstrate that ischemic injury promotes the association of death-associated protein kinase 1 with the NMDA receptor, thereby potentiating its activity, and show that disrupting this association reduces damage to the brain.
Authors:
Henry G S Martin; Yu Tian Wang
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Publication Detail:
Type:  Comment; Journal Article    
Journal Detail:
Title:  Cell     Volume:  140     ISSN:  1097-4172     ISO Abbreviation:  Cell     Publication Date:  2010 Jan 
Date Detail:
Created Date:  2010-02-09     Completed Date:  2010-02-25     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0413066     Medline TA:  Cell     Country:  United States    
Other Details:
Languages:  eng     Pagination:  174-6     Citation Subset:  IM    
Copyright Information:
Copyright 2010 Elsevier Inc. All rights reserved.
Affiliation:
Brain Research Centre, Vancouver Coastal Health Research Institute and University of British Columbia, 2211 Wesbrook Mall, Vancouver, BC V6T 2B5, Canada.
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MeSH Terms
Descriptor/Qualifier:
Animals
Apoptosis Regulatory Proteins / antagonists & inhibitors,  metabolism*
Brain / metabolism,  pathology
Brain Ischemia / metabolism,  pathology
Calcium-Calmodulin-Dependent Protein Kinases / antagonists & inhibitors,  metabolism*
Peptides / metabolism
Receptors, N-Methyl-D-Aspartate / metabolism*
Stroke / drug therapy,  metabolism*,  pathology
Chemical
Reg. No./Substance:
0/Apoptosis Regulatory Proteins; 0/NR2B NMDA receptor; 0/Peptides; 0/Receptors, N-Methyl-D-Aspartate; EC 2.7.11.1/death-associated protein kinase; EC 2.7.11.17/Calcium-Calmodulin-Dependent Protein Kinases
Comments/Corrections
Comment On:
Cell. 2010 Jan 22;140(2):222-34   [PMID:  20141836 ]

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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