Document Detail


Bladder selectivity of the novel β₃-agonist ritobegron (KUC-7483) explored by in vitro and in vivo studies in the rat.
MedLine Citation:
PMID:  22552730     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
We performed in vitro and in vivo experiments to evaluate the pharmacological profile of ritobegron and its effects on the bladder in rats. β(3)-AR selectivity was assessed using CHO cells expressing various subtypes of the human β-adrenoceptor (AR). Effects on isolated organs were evaluated using the organ-bath method. Effects on intravesical pressure, heart rate, and mean blood pressure were evaluated in urethane-anesthetized rats. Ritobegron increased cAMP accumulation in a concentration-dependent manner in CHO cells expressing any one of three human β-AR, its selectivity for β(3)-AR being 301-fold and 32-fold higher versus β(1)-AR and β(2)-AR, respectively. Ritobegron decreased the resting tension of the isolated bladder in a concentration-dependent manner (EC(50), 7.7 × 10(-8) mol/L; maximal relaxation, 97.0 %), and the β(3)-AR antagonist SR58894A produced a parallel rightward-shift of this concentration-response curve without altering the maximal response [pK(B) value, 6.43]. Ritobegron concentration-dependently increased atrial rate and decreased myometrial contractions in vitro, and its selectivity for the bladder was 2,078-fold higher versus the atria and 14-fold higher versus the uterus. In vivo, ritobegron induced a dose-dependent decrease in intravesical pressure (ED(50) 0.4 mg/kg), without affecting heart rate and only slightly lowering mean blood pressure. Thus, ritobegron displayed potent and selective β(3)-AR agonistic activity toward transfected human β-AR and exhibited a high selectivity for the bladder versus other organs in rats. Moreover, it decreased intravesical pressure with minimal effects on the cardiovascular system in anesthetized rats. These results suggest that ritobegron shows promise as a potential agent for the treatment of overactive bladder.
Authors:
Itaru Maruyama; Yoshiaki Goi; Satoshi Tatemichi; Kazuyasu Maruyama; Yuji Hoyano; Yoshinobu Yamazaki; Hiroshi Kusama
Publication Detail:
Type:  In Vitro; Journal Article     Date:  2012-05-03
Journal Detail:
Title:  Naunyn-Schmiedeberg's archives of pharmacology     Volume:  385     ISSN:  1432-1912     ISO Abbreviation:  Naunyn Schmiedebergs Arch. Pharmacol.     Publication Date:  2012 Aug 
Date Detail:
Created Date:  2012-07-16     Completed Date:  2012-11-26     Revised Date:  2012-12-21    
Medline Journal Info:
Nlm Unique ID:  0326264     Medline TA:  Naunyn Schmiedebergs Arch Pharmacol     Country:  Germany    
Other Details:
Languages:  eng     Pagination:  845-52     Citation Subset:  IM    
Affiliation:
Central Research Laboratory, Kissei Pharmaceutical Co., Ltd., Nagano, Japan. itaru_maruyama@pharm.kissei.co.jp
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MeSH Terms
Descriptor/Qualifier:
Acetates / pharmacology*
Adrenergic beta-3 Receptor Agonists / pharmacology*
Adrenergic beta-3 Receptor Antagonists / pharmacology
Adrenergic beta-Agonists / pharmacology
Animals
CHO Cells
Cricetinae
Cricetulus
Cyclic AMP / metabolism
Female
Forskolin / pharmacology
Heart Rate / drug effects
Humans
Isoproterenol / pharmacology
Male
Muscle Relaxation / drug effects
Organ Specificity
Rats
Rats, Sprague-Dawley
Substrate Specificity
Urinary Bladder / drug effects*
Urinary Bladder, Overactive / drug therapy
p-Hydroxyamphetamine / analogs & derivatives*,  pharmacology
Chemical
Reg. No./Substance:
0/Acetates; 0/Adrenergic beta-3 Receptor Agonists; 0/Adrenergic beta-3 Receptor Antagonists; 0/Adrenergic beta-Agonists; 0/ritobegron ethyl; 103-86-6/p-Hydroxyamphetamine; 60-92-4/Cyclic AMP; 66428-89-5/Forskolin; 7683-59-2/Isoproterenol

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