Document Detail


Bilateral thalamic lesions in a newborn with intrauterine asphyxia after maternal cardiac arrest--a case report with literature review.
MedLine Citation:
PMID:  11593379     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Hypoxic-ischemic brain damage in preterm and term infants is one major cause of neonatal neurologic morbidity. Depending on the gestational age and the extent of hypoxia, different pathologic findings have been observed. Hypoxic-ischemic lesion of the thalamus is the least common form of cerebral injury. Although long-term outcome with spastic or extrapyramidal cerebral palsy is known, clinical features in the neonatal period are not well described. We report an infant with bilateral hypoxic-ischemic thalamic lesions after maternal cardiac arrest at 28 weeks of gestation. Clinical features and diagnostic results of our patient are compared to information given in the literature to define the clinical entity of hypoxic-ischemic thalamic lesions in neonates better.
Authors:
M C Banerjea; C P Speer
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Publication Detail:
Type:  Case Reports; Journal Article    
Journal Detail:
Title:  Journal of perinatology : official journal of the California Perinatal Association     Volume:  21     ISSN:  0743-8346     ISO Abbreviation:  J Perinatol     Publication Date:  2001 Sep 
Date Detail:
Created Date:  2001-10-10     Completed Date:  2001-11-01     Revised Date:  2009-11-03    
Medline Journal Info:
Nlm Unique ID:  8501884     Medline TA:  J Perinatol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  405-9     Citation Subset:  IM    
Affiliation:
Childrens' Hospital, Department of Neonatology, University of Wuerzburg, Wuerzburg, Germany.
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MeSH Terms
Descriptor/Qualifier:
Adult
Female
Fetal Hypoxia / complications*
Heart Arrest*
Humans
Infant, Newborn
Magnetic Resonance Imaging
Pregnancy
Pregnancy Complications, Cardiovascular*
Thalamic Diseases / etiology*,  ultrasonography

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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