Document Detail


Beyond SHM and CSR: AID and related cytidine deaminases in the host response to viral infection.
MedLine Citation:
PMID:  17560276     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
As the primary effector of immunoglobulin somatic hypermutation (SHM) and class switch recombination (CSR), activation-induced cytidine deaminase (AID) serves an important function in the adaptive immune response. Recent advances have demonstrated that AID and a group of closely related cytidine deaminases, the APOBEC3 proteins, also act in the innate host response to viral infection. Antiviral activity was first attributed to APOBEC3G as a potent inhibitor of HIV. It is now apparent that the targets of the APOBEC3 proteins extend beyond HIV, with family members acting against a wide variety of viruses as well as host-encoded retrotransposable genetic elements. Although it appears to function through a different mechanism, AID also possesses antiviral properties. Independent of its antibody diversification functions, AID protects against transformation by Abelson murine leukemia virus (Ab-MLV), an oncogenic retrovirus. Additionally, AID has been implicated in the host response to other pathogenic viruses. These emerging roles for the AID/APOBEC cytidine deaminases in viral infection suggest an intriguing evolutionary connection of innate and adaptive immune mechanisms.
Authors:
Brad R Rosenberg; F Nina Papavasiliou
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Review    
Journal Detail:
Title:  Advances in immunology     Volume:  94     ISSN:  0065-2776     ISO Abbreviation:  Adv. Immunol.     Publication Date:  2007  
Date Detail:
Created Date:  2007-06-11     Completed Date:  2007-09-12     Revised Date:  2007-12-03    
Medline Journal Info:
Nlm Unique ID:  0370425     Medline TA:  Adv Immunol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  215-44     Citation Subset:  IM    
Affiliation:
Laboratory of Lymphocyte Biology, The Rockefeller University, New York, New York, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Cytidine Deaminase / immunology*
HIV Infections / immunology
Humans
Immunoglobulin Class Switching
Somatic Hypermutation, Immunoglobulin
Virus Diseases / immunology*
Grant Support
ID/Acronym/Agency:
AI071029/AI/NIAID NIH HHS; GM07739/GM/NIGMS NIH HHS
Chemical
Reg. No./Substance:
EC 3.5.4.-/AICDA (activation-induced cytidine deaminase); EC 3.5.4.5/Cytidine Deaminase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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