Document Detail


Baby on board: do responses to stress in the maternal brain mediate adverse pregnancy outcome?
MedLine Citation:
PMID:  20546772     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Stress and adverse environmental surroundings result in suboptimal conditions in a pregnant mother such that she may experience poor pregnancy outcome including complete pregnancy failure and preterm labor. Furthermore her developing baby is at risk of adverse programming, which confers susceptibility to long term ill health. While some mechanisms at the feto-maternal interface underlying these conditions are understood, the underlying cause for their adverse adaptation is often not clear. Progesterone plays a key role at many levels, including control of neuroendocrine responses to stress, procuring the required immune balance and controlling placental and decidual function, and lack of progesterone can explain many of the unwanted consequences of stress. How stress that is perceived by the mother inhibits progesterone secretion and action is beginning to be investigated. This overview of maternal neuroendocrine responses to stress throughout pregnancy analyses how they interact to compromise progesterone secretion and precipitate undesirable effects in mother and offspring.
Authors:
Alison J Douglas
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Review     Date:  2010-05-31
Journal Detail:
Title:  Frontiers in neuroendocrinology     Volume:  31     ISSN:  1095-6808     ISO Abbreviation:  Front Neuroendocrinol     Publication Date:  2010 Jul 
Date Detail:
Created Date:  2010-07-26     Completed Date:  2010-11-05     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  7513292     Medline TA:  Front Neuroendocrinol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  359-76     Citation Subset:  IM    
Copyright Information:
Copyright 2010 Elsevier Inc. All rights reserved.
Affiliation:
Laboratory of Neuroendocrinology, Centre for Integrative Physiology, University of Edinburgh, Hugh Robson Building, George Square, Edinburgh EH8 9XD, United Kingdom. alison.j.douglas@ed.ac.uk
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MeSH Terms
Descriptor/Qualifier:
Animals
Brain / metabolism,  physiopathology*
Female
Humans
Infant, Newborn
Models, Biological
Mothers / psychology
Neurosecretory Systems / metabolism,  physiology
Pregnancy
Pregnancy Outcome*
Prenatal Exposure Delayed Effects / metabolism,  physiopathology*
Stress, Psychological / metabolism,  physiopathology*
Grant Support
ID/Acronym/Agency:
//Medical Research Council; //Wellcome Trust

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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