Document Detail


BCR-induced superoxide negatively regulates B-cell proliferation and T-cell-independent type 2 Ab responses.
MedLine Citation:
PMID:  19877015     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Superoxide and its derivatives have been implicated as secondary messenger molecules that influence signaling cascades in non-phagocytes. B lymphocytes produce superoxide after BCR ligation. We found that these ROS regulate B-cell signaling and entry into the cell cycle. B cells from mice deficient in the gp91(phox) subunit of the NADPH oxidase complex are unable to generate ROS after BCR ligation. However, after BCR stimulation, more gp91(phox) KO B cells enter the G1 stage of the cell cycle and proliferate than WT B cells. BCR ligation leads to a more rapid decrease in p27(Kip1) levels in gp91(phox) KO B cells. Gp91(phox) KO mice display enhanced T-cell-independent type 2, but normal T-dependent Ab responses. ROS-dependent regulation of BCR-induced proliferation may help modulate the size of the humoral response to T-cell-independent type 2 Ag immunization.
Authors:
Sabrina M Richards; Edward A Clark
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  European journal of immunology     Volume:  39     ISSN:  1521-4141     ISO Abbreviation:  Eur. J. Immunol.     Publication Date:  2009 Dec 
Date Detail:
Created Date:  2009-12-09     Completed Date:  2010-01-06     Revised Date:  2011-05-12    
Medline Journal Info:
Nlm Unique ID:  1273201     Medline TA:  Eur J Immunol     Country:  Germany    
Other Details:
Languages:  eng     Pagination:  3395-403     Citation Subset:  IM    
Affiliation:
Department of Immunology, University of Washington, Seattle, WA 98195, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
B-Lymphocytes / cytology,  metabolism*
Blotting, Western
Calcium / metabolism
Cell Cycle
Cell Proliferation*
Flow Cytometry
G1 Phase
Immunoglobulin G / metabolism*
Membrane Glycoproteins / genetics,  metabolism
Mice
Mice, Inbred C57BL
Mice, Knockout
NADPH Oxidase / genetics,  metabolism
Reactive Oxygen Species / metabolism
Receptors, Antigen, B-Cell / physiology*
Superoxides / metabolism*
T-Lymphocytes / cytology,  metabolism
Grant Support
ID/Acronym/Agency:
DE 16381/DE/NIDCR NIH HHS; GM37905/GM/NIGMS NIH HHS; R01 DE016381-05/DE/NIDCR NIH HHS; R01 GM037905-20/GM/NIGMS NIH HHS
Chemical
Reg. No./Substance:
0/Immunoglobulin G; 0/Membrane Glycoproteins; 0/Reactive Oxygen Species; 0/Receptors, Antigen, B-Cell; 11062-77-4/Superoxides; 7440-70-2/Calcium; EC 1.6.3.1/Cybb protein, mouse; EC 1.6.3.1/NADPH Oxidase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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