Document Detail


BAD, a proapoptotic member of the BCL2 family, is a potential therapeutic target in hepatocellular carcinoma.
MedLine Citation:
PMID:  20647330     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Proteins of the BCL2 family are key regulators of apoptosis. Their expression levels are frequently altered in cancers, enabling tumor cells to survive. To gain insight into the pathogenesis of hepatocellular carcinoma (HCC), we performed a comprehensive survey of the expression of the members of the BCL2 family in samples obtained from surgically resected HCCs. Here, we report the occurrence of a new molecular anomaly, consisting of a strong reduction in the expression of the proapoptotic protein BAD in HCC compared with surrounding nontumoral tissue. We investigate the function of BAD in a panel of HCC cell lines. Using gene overexpression and RNA interference, we show that BAD is involved in the cytotoxic effects of sorafenib, a multikinase blocker, which is currently the sole therapeutic drug effective for the treatment of HCC. Finally, we report that ABT-737, a compound that interacts with proteins of the BCL2 family and exhibits a BAD-like reactivity, sensitizes HCC cells toward sorafenib-induced apoptosis. Collectively, our findings indicate that BAD is a key regulator of apoptosis in HCC and an important determinant of HCC cell response to sorafenib.
Authors:
Antoine Galmiche; Zakaria Ezzoukhry; Catherine François; Christophe Louandre; Charles Sabbagh; Eric Nguyen-Khac; Véronique Descamps; Nathalie Trouillet; Corinne Godin; Jean-Marc Regimbeau; Jean-Paul Joly; Jean-Claude Barbare; Gilles Duverlie; Jean-Claude Mazière; Denis Chatelain
Publication Detail:
Type:  Comparative Study; Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-07-20
Journal Detail:
Title:  Molecular cancer research : MCR     Volume:  8     ISSN:  1557-3125     ISO Abbreviation:  Mol. Cancer Res.     Publication Date:  2010 Aug 
Date Detail:
Created Date:  2010-08-18     Completed Date:  2010-12-02     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101150042     Medline TA:  Mol Cancer Res     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1116-25     Citation Subset:  IM    
Affiliation:
Laboratoire de Biochimie, CHU Nord, Amiens Cedex, France. Galmiche.Antoine@chu-amiens.fr
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MeSH Terms
Descriptor/Qualifier:
Antineoplastic Agents / pharmacology
Apoptosis / drug effects*
Benzenesulfonates / pharmacology
Biphenyl Compounds / pharmacology
Blotting, Western
Carcinoma, Hepatocellular / drug therapy,  metabolism*,  pathology*
Cell Line, Tumor
Cell Proliferation / drug effects
Drug Synergism
Humans
Liver / drug effects,  metabolism,  pathology
Liver Neoplasms / drug therapy,  metabolism*,  pathology*
Nitrophenols / pharmacology
Piperazines / pharmacology
Proto-Oncogene Proteins c-bcl-2 / metabolism*
Pyridines / pharmacology
RNA, Messenger / genetics
Reverse Transcriptase Polymerase Chain Reaction
Sulfonamides / pharmacology
Tumor Markers, Biological / metabolism
Chemical
Reg. No./Substance:
0/ABT-737; 0/Antineoplastic Agents; 0/Benzenesulfonates; 0/Biphenyl Compounds; 0/Nitrophenols; 0/Piperazines; 0/Proto-Oncogene Proteins c-bcl-2; 0/Pyridines; 0/RNA, Messenger; 0/Sulfonamides; 0/Tumor Markers, Biological; 0/sorafenib

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