Document Detail


B cell-specific expression of B7-2 is required for follicular Th cell function in response to vaccinia virus.
MedLine Citation:
PMID:  21441451     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Follicular Th (T(FH)) cells are specialized in provision of help to B cells that is essential for promoting protective Ab responses. CD28/B7 (B7-1 and B7-2) interactions are required for germinal center (GC) formation, but it is not clear if they simply support activation of naive CD4 T cells during initiation of responses by dendritic cells or if they directly control T(FH) cells and/or directly influence follicular B cell differentiation. Using a model of vaccinia virus infection, we show that B7-2 but not B7-1 deficiency profoundly impaired T(FH) cell development but did not affect CD4 T cell priming and Th1 differentiation. Consistent with this, B7-2 but not B7-1 was required for acquisition of GC B cell phenotype, plasma cell generation, and virus-specific neutralizing Ab responses. Mixed adoptive transfer experiments indicated that bidirectional interactions between CD28 expressed on activated T cells and B7-2 expressed on follicular B cells were essential for maintenance of the T(FH) phenotype and GC B cell development. Our data provide new insight into the source and nature of molecules required for T(FH) cells to direct GC B cell responses.
Authors:
Samira Salek-Ardakani; Youn Soo Choi; Mohammed Rafii-El-Idrissi Benhnia; Rachel Flynn; Ramon Arens; Stephen Shoenberger; Shane Crotty; Michael Croft; Shahram Salek-Ardakani
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2011-03-25
Journal Detail:
Title:  Journal of immunology (Baltimore, Md. : 1950)     Volume:  186     ISSN:  1550-6606     ISO Abbreviation:  J. Immunol.     Publication Date:  2011 May 
Date Detail:
Created Date:  2011-04-20     Completed Date:  2011-07-05     Revised Date:  2012-05-02    
Medline Journal Info:
Nlm Unique ID:  2985117R     Medline TA:  J Immunol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  5294-303     Citation Subset:  AIM; IM    
Affiliation:
Division of Immune Regulation, La Jolla Institute for Allergy and Immunology, San Diego, CA 92037, USA.
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MeSH Terms
Descriptor/Qualifier:
Adoptive Transfer
Animals
Antigens, CD28 / biosynthesis,  immunology
Antigens, CD86 / biosynthesis,  immunology*
B-Lymphocytes / cytology,  immunology*,  metabolism
Cell Differentiation / immunology
Cell Separation
Female
Flow Cytometry
Germinal Center / cytology,  immunology
Lymphocyte Activation / immunology*
Mice
Mice, Inbred C57BL
Mice, Knockout
Plasma Cells / cytology,  immunology
Spleen / cytology,  immunology
T-Lymphocytes, Helper-Inducer / immunology*
Vaccinia / immunology*
Vaccinia virus / immunology
Grant Support
ID/Acronym/Agency:
AI63107/AI/NIAID NIH HHS; AI67341/AI/NIAID NIH HHS; AI72543/AI/NIAID NIH HHS; AI77079/AI/NIAID NIH HHS; CA91837/CA/NCI NIH HHS; R01 AI063107-07/AI/NIAID NIH HHS; R01 AI067341-05/AI/NIAID NIH HHS; R01 AI072543-05/AI/NIAID NIH HHS; R01 CA091837-09/CA/NCI NIH HHS; R21 AI077079-02/AI/NIAID NIH HHS
Chemical
Reg. No./Substance:
0/Antigens, CD28; 0/Antigens, CD86

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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