Document Detail


Axonal damage in multiple sclerosis.
MedLine Citation:
PMID:  21425267     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Multiple sclerosis is a debilitating disease of the central nervous system that has been characteristically classified as an immune-mediated destruction of myelin, the protective coating on nerve fibers. Although the mechanisms responsible for the immune attack to central nervous system myelin have been the subject of intense investigation, more recent studies have focused on the neurodegenerative component, which is cause of clinical disability in young adults and appears to be only partially controlled by immunomodulatory therapies. Here, we review distinct, but not mutually exclusive, mechanisms of pathogenesis of axonal damage in multiple sclerosis patients that are either consequent to long-term demyelination or independent from it. We propose that the complexity of axonal degeneration and the heterogeneity of the underlying pathogenetic mechanisms should be taken into consideration for the design of targeted therapeutic intervention.
Authors:
Jeffery D Haines; Matilde Inglese; Patrizia Casaccia
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Publication Detail:
Type:  Journal Article; Research Support, American Recovery and Reinvestment Act; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  The Mount Sinai journal of medicine, New York     Volume:  78     ISSN:  1931-7581     ISO Abbreviation:  Mt. Sinai J. Med.     Publication Date:    2011 Mar-Apr
Date Detail:
Created Date:  2011-03-22     Completed Date:  2011-07-27     Revised Date:  2012-05-22    
Medline Journal Info:
Nlm Unique ID:  0241032     Medline TA:  Mt Sinai J Med     Country:  United States    
Other Details:
Languages:  eng     Pagination:  231-43     Citation Subset:  IM    
Copyright Information:
© 2011 Mount Sinai School of Medicine.
Affiliation:
Departments of Neuroscience Neurology and Genetics and Genomics, Mount Sinai School of Medicine, New York, NY, USA.
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MeSH Terms
Descriptor/Qualifier:
Activities of Daily Living
Adult
Animals
Aspartic Acid / analogs & derivatives,  metabolism
Axons* / metabolism,  pathology
Humans
Immunity / drug effects
Immunologic Factors* / metabolism,  therapeutic use
Inflammation / immunology,  physiopathology
Multiple Sclerosis* / metabolism,  pathology,  physiopathology,  therapy
Myelin Sheath* / metabolism,  pathology
Nerve Degeneration* / metabolism,  pathology
Nervous System Autoimmune Disease, Experimental / metabolism,  pathology,  physiopathology,  therapy
Positron-Emission Tomography
Grant Support
ID/Acronym/Agency:
NS42925-08/NS/NINDS NIH HHS; R01 NS042925/NS/NINDS NIH HHS; R01 NS042925-07S1/NS/NINDS NIH HHS; R01 NS042925-08/NS/NINDS NIH HHS; R01 NS042925-09/NS/NINDS NIH HHS; R01-NS42925-07S1/NS/NINDS NIH HHS
Chemical
Reg. No./Substance:
0/Immunologic Factors; 56-84-8/Aspartic Acid; 997-55-7/N-acetylaspartate

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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