Document Detail


Autophagy for tissue homeostasis and neuroprotection.
MedLine Citation:
PMID:  21030235     Owner:  NLM     Status:  In-Data-Review    
Abstract/OtherAbstract:
Although autophagy has frequently been viewed as a cell death mechanism in the mammalian system, it is now considered as indispensable for the homeostasis of cells, tissues, and organisms. Basal or stress-induced autophagy plays essential and diverse roles in a variety of tissues, due to its cytoprotective properties. In this review, we briefly discuss the different homeostatic functions of autophagy that have been finely dissected in mammals through the generation and characterization of animal models with tissue-specific autophagic alterations. In addition, and given the importance of constitutive autophagy in neuronal tissues, we describe in more detail the specific roles of autophagy in the central nervous system (CNS). Finally, we discuss the contribution of autophagy malfunctions to the development of several common neurological disorders and the potential benefits of pharmacologically induced autophagy for the avoidance of neurodegeneration.
Authors:
Guillermo Mariño; Frank Madeo; Guido Kroemer
Related Documents :
15953815 - Oxidative stress and neuronal death/survival signaling in cerebral ischemia.
15275405 - Programmed t-cell death in experimental chagas disease.
19170195 - Apoptosis induction by sulfur-containing compounds in malignant and nonmalignant human ...
18079135 - The hypersensitive response; the centenary is upon us but how much do we know?
1832565 - Novel expression pattern of a new member of the mip-1 family of cytokine-like genes.
15668915 - A new carboxamide compound exerts immuno-suppressive activity by inhibiting dendritic c...
Publication Detail:
Type:  Journal Article     Date:  2010-10-26
Journal Detail:
Title:  Current opinion in cell biology     Volume:  23     ISSN:  1879-0410     ISO Abbreviation:  Curr. Opin. Cell Biol.     Publication Date:  2011 Apr 
Date Detail:
Created Date:  2011-04-15     Completed Date:  -     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8913428     Medline TA:  Curr Opin Cell Biol     Country:  England    
Other Details:
Languages:  eng     Pagination:  198-206     Citation Subset:  IM    
Copyright Information:
Copyright © 2010 Elsevier Ltd. All rights reserved.
Affiliation:
INSERM, U848, F-94805 Villejuif, France; Institut Gustave Roussy, F-94805 Villejuif, France; Université Paris Sud, Paris 11, F-94805 Villejuif, France.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Functional interactions between nucleoporins and chromatin.
Next Document:  The ever-expanding list of HLA alleles: changing HLA nomenclature and its relevance to clinical tran...