| Atypical hemolytic uremic syndrome: telling the difference between H and Y. | |
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MedLine Citation:
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PMID: 20877372 Owner: NLM Status: In-Process |
Abstract/OtherAbstract:
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Mutations in the complement factor H (CFH) gene are frequently associated with atypical hemolytic uremic syndrome (aHUS). Hakobyan et al. have developed novel reagents that can rapidly determine the contribution of each CFH allele to the total plasma CFH pool, showing that low-expression CFH alleles are important risk factors for the development of aHUS. These reagents represent a significant contribution to the techniques used to determine susceptibility factors among individuals with aHUS. |
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Authors:
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E Goicoechea de Jorge; Matthew C Pickering |
Publication Detail:
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Type: Comment; Journal Article; Research Support, Non-U.S. Gov't |
Journal Detail:
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Title: Kidney international Volume: 78 ISSN: 1523-1755 ISO Abbreviation: Kidney Int. Publication Date: 2010 Oct |
Date Detail:
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Created Date: 2010-09-29 Completed Date: - Revised Date: - |
Medline Journal Info:
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Nlm Unique ID: 0323470 Medline TA: Kidney Int Country: United States |
Other Details:
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Languages: eng Pagination: 721-3 Citation Subset: IM |
Affiliation:
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Centre for Complement and Inflammation Research, Division of Immunology and Inflammation, Faculty of Medicine, Imperial College, London, UK. |
Export Citation:
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APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
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| Grant Support | |
ID/Acronym/Agency:
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WT082291MA//Wellcome Trust |
| Comments/Corrections | |
Comment On:
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Kidney Int. 2010 Oct;78(8):782-8
[PMID:
20703214
]
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From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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