Document Detail

The Athlete's Heart vs. the Failing Heart: Can Signaling Explain the Two Distinct Outcomes?
MedLine Citation:
PMID:  21487028     Owner:  NLM     Status:  In-Data-Review    
Cardiac remodeling is typically associated with disease and can lead to heart failure. In contrast, remodeling that occurs in the athlete's heart is considered an adaptive physiological response. This review provides an overview of signaling mechanisms responsible for inducing left ventricular hypertrophy in the athlete's heart and in settings of pathological hypertrophy and heart failure.
Kate L Weeks; Julie R McMullen
Related Documents :
10381288 - Correlation between coronary morphology and molecular markers of fibrinolysis in unstab...
3872968 - Determinants of operative mortality and long-term survival in patients with unstable an...
10384828 - Characterization of the unstable lesion by angiography, angioscopy, and intravascular u...
25181728 - Cardiomyopathy induced by sinus tachycardia in combat wounded: a case study.
24118198 - Clinical and angiographic outcomes with everolimus eluting stents for the treatment of ...
2745518 - The effects of activated complement on myocardial performance in vitro.
Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Physiology (Bethesda, Md.)     Volume:  26     ISSN:  1548-9221     ISO Abbreviation:  Physiology (Bethesda)     Publication Date:  2011 Apr 
Date Detail:
Created Date:  2011-04-13     Completed Date:  -     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101208185     Medline TA:  Physiology (Bethesda)     Country:  United States    
Other Details:
Languages:  eng     Pagination:  97-105     Citation Subset:  IM    
1Cardiac Hypertrophy Laboratory, Baker IDI Heart and Diabetes Institute, and.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine

Previous Document:  mTor signaling in skeletal muscle during sepsis and inflammation: where does it all go wrong?
Next Document:  Heterogeneous cardiac proteasomes: mandated by diverse substrates?