Document Detail


Astrocytes contacting HIV-1-infected macrophages increase the release of CCL2 in response to the HIV-1-dependent enhancement of membrane-associated TNFα in macrophages.
MedLine Citation:
PMID:  20737475     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The presence of human immunodeficiency virus (HIV)-infected macrophages in the parenchyma of central nervous system is an hallmark of acquired immunodeficiency syndrome-related neuroinflammation. Once penetrated the blood-brain barrier (BBB), macrophages closely interact with astrocytes, beginning with those lying beneath the BBB endothelium. By investigating the consequences of the cell-cell interaction between HIV-infected macrophages and astrocytes, we observed that the HIV-1 expression in macrophagic cells correlated with increased chemotactic activity in supernatants of astroglial cells. Gene array analysis revealed an impressive increase in the transcription of the gene for the CCL2/MCP-1 chemokine in astroglial cells isolated from HIV-1-infected co-cultures compared with cells from uninfected co-cultures. This phenomenon coupled with the increase in CCL2 release and depended on the cell-cell contact. In addition, it was a consequence of the HIV-1-induced enhancement of membrane-associated tumor necrosis factor-α in macrophagic cells, and correlated with increased levels of nuclear factor kappaB activation in astroglial cells. These observations could mirror a mechanism of recruitment of leukocytes through the BBB, likely contributing to the increase in both viral load and inflammation in central nervous system of HIV-infected patients.
Authors:
Claudia Muratori; Giorgio Mangino; Elisabetta Affabris; Maurizio Federico
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Glia     Volume:  58     ISSN:  1098-1136     ISO Abbreviation:  Glia     Publication Date:  2010 Dec 
Date Detail:
Created Date:  2010-10-20     Completed Date:  2011-01-06     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8806785     Medline TA:  Glia     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1893-904     Citation Subset:  IM    
Affiliation:
National AIDS Center, Istituto Superiore di Sanità, Rome, Italy.
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MeSH Terms
Descriptor/Qualifier:
Astrocytes / immunology,  pathology,  virology*
Astrocytoma
Brain Neoplasms
Cell Communication / immunology
Chemokine CCL2 / genetics,  immunology*
Chemotaxis / immunology
Coculture Techniques
Gene Expression / immunology
HIV Infections / immunology*,  pathology
HIV-1*
Humans
I-kappa B Proteins / metabolism
Macrophages / immunology,  pathology,  virology*
Membrane Proteins / immunology,  metabolism
Oligonucleotide Array Sequence Analysis
Tumor Necrosis Factor-alpha / immunology*,  metabolism
U937 Cells
Chemical
Reg. No./Substance:
0/CCL2 protein, human; 0/Chemokine CCL2; 0/I-kappa B Proteins; 0/Membrane Proteins; 0/Tumor Necrosis Factor-alpha; 139874-52-5/NF-kappaB inhibitor alpha

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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