Document Detail


Association of a novel functional promoter variant (rs2075533 C>T) in the apoptosis gene TNFSF8 with risk of lung cancer--a finding from Texas lung cancer genome-wide association study.
MedLine Citation:
PMID:  21292647     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Published genome-wide association studies (GWASs) have identified few variants in the known biological pathways involved in lung cancer etiology. To mine the possibly hidden causal single nucleotide polymorphisms (SNPs), we explored all SNPs in the extrinsic apoptosis pathway from our published GWAS dataset for 1154 lung cancer cases and 1137 cancer-free controls. In an initial association analysis of 611 tagSNPs in 41 apoptosis-related genes, we identified only 10 tagSNPs associated with lung cancer risk with a P value<10(-2), including four tagSNPs in DAPK1 and three tagSNPs in TNFSF8. Unlike DAPK1 SNPs, TNFSF8 rs2181033 tagged other four predicted functional but untyped SNPs (rs776576, rs776577, rs31813148 and rs2075533) in the promoter region. Therefore, we further tested binding affinity of these four SNPs by performing the electrophoretic mobility shift assay. We found that only rs2075533T allele modified levels of nuclear proteins bound to DNA, leading to significantly decreased expression of luciferase reporter constructs by 5- to -10-fold in H1299, HeLa and HCT116 cell lines compared with the C allele. We also performed a replication study of the untyped rs2075533 in an independent Texas population but did not confirm the protective effect. We further performed a mini meta-analysis for SNPs of TNFSF8 obtained from other four published lung cancer GWASs with 12  214 cases and 47  721 controls, and we found that only rs3181366 (r2=0.69 with the untyped rs2075533) was associated to lung cancer risk (P=0.008). Our findings suggest a possible role of novel TNFSF8 variants in susceptibility to lung cancer.
Authors:
Sheng Wei; Jiangong Niu; Hui Zhao; Zhensheng Liu; Li-E Wang; Younghun Han; Wei V Chen; Christopher I Amos; Thorunn Rafnar; Patrick Sulem; Kari Stefansson; Maria T Landi; Neil E Caporaso; Demetrius Albanes; Michael J Thun; James D McKay; Paul Brennan; Yufei Wang; Richard S Houlston; Margaret R Spitz; Qingyi Wei
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural     Date:  2011-02-02
Journal Detail:
Title:  Carcinogenesis     Volume:  32     ISSN:  1460-2180     ISO Abbreviation:  Carcinogenesis     Publication Date:  2011 Apr 
Date Detail:
Created Date:  2011-03-31     Completed Date:  2011-06-09     Revised Date:  2012-10-09    
Medline Journal Info:
Nlm Unique ID:  8008055     Medline TA:  Carcinogenesis     Country:  England    
Other Details:
Languages:  eng     Pagination:  507-15     Citation Subset:  IM    
Affiliation:
Department of Epidemiology, The University of Texas M. D. Anderson Cancer Center, Houston, Texas 77030, USA.
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MeSH Terms
Descriptor/Qualifier:
Adult
Aged
Antigens, CD30 / physiology
Apoptosis
CD30 Ligand / genetics*
Cell Line, Tumor
Female
Genome-Wide Association Study*
Humans
Lung Neoplasms / etiology,  genetics*
Male
Middle Aged
Polymorphism, Single Nucleotide*
Promoter Regions, Genetic*
Risk
Texas
Grant Support
ID/Acronym/Agency:
P30 CA016672/CA/NCI NIH HHS; R01 CA121197/CA/NCI NIH HHS; R01 CA133996/CA/NCI NIH HHS; R01CA055769/CA/NCI NIH HHS; R01CA121197/CA/NCI NIH HHS; R01CA127219/CA/NCI NIH HHS; R01CA131274/CA/NCI NIH HHS; R01ES011740/ES/NIEHS NIH HHS; U19 CA148127/CA/NCI NIH HHS
Chemical
Reg. No./Substance:
0/Antigens, CD30; 0/CD30 Ligand; 0/TNFSF8 protein, human
Comments/Corrections

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