Document Detail


Association between the angiotensin-converting enzyme gene polymorphisms and tissue oxygenation during exercise in patients with COPD.
MedLine Citation:
PMID:  11888948     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
STUDY OBJECTIVES: We have recently determined that the angiotensin-converting enzyme (ACE) DD genotype might be associated with pulmonary hypertension during exercise in patients with COPD. Therefore, this study was designed to determine whether ACE gene polymorphisms adversely affect tissue oxygenation during exercise in patients with COPD. DESIGN: Cross-sectional analysis. SETTING: University hospital. PATIENTS: Thirty-nine patients (14 patients with II genotype, 12 patients with ID genotype, and 13 patients with DD genotype). INTERVENTIONS: All patients underwent right-heart catheterization and constant-load exercise testing for 5 min on an ergometer. Measurements and results: The ratio of the change in oxygen delivery (DO(2)) to the increase in oxygen consumption (O(2)) during exercise (DeltaDO(2)/DeltaO(2)) was significantly lower in patients with the DD genotype (1.5 +/- 0.2) than in those with the II genotype (1.9 +/- 0.3, p = 0.0006) and the ID genotype (1.7 +/- 0.2, p = 0.037). Mixed venous oxygen tension (PO(2)) after exercise in patients with the DD genotype (23.5 +/- 1.5 mm Hg) was also significantly lower than in patients with the II genotype (26.7 +/- 1.6 mm Hg, p = 0.0002) and the ID genotype (25.0 plus minus 2.0 mm Hg, p = 0.045). In addition, the change in plasma concentration of lactate during exercise (DeltaLactate) was significantly higher in patients with DD genotype (33.3 +/- 4.3 mmol/L) than in those with the II genotype (25.5 +/- 3.6 mmol/L, p = 0.0002) and the ID genotype (28.8 +/- 4.0 mmol/L, p = 0.029). The mean pulmonary arterial pressure after exercise was significantly correlated with DeltaDO(2)/DeltaO(2) (r = - 0.423, p = 0.0076) but not with PvO(2) after exercise and with DeltaLactate. CONCLUSIONS: The ACE DD genotype may be associated with an impairment in peripheral tissue oxygenation during exercise in patients with COPD.
Authors:
Hiroshi Kanazawa; Toshihiro Otsuka; Kazuto Hirata; Junichi Yoshikawa
Related Documents :
1995228 - Right ventricular dysfunction in chronic obstructive pulmonary disease. evaluation and ...
11587988 - Exhaled nitric oxide and hydrogen peroxide in patients with chronic obstructive pulmona...
22892548 - Lifestyle factors predicting changes in aerobic capacity of aging firefighters at 3- an...
12761008 - Prone position improves lung mechanical behavior and enhances gas exchange efficiency i...
8047578 - Aftereffects of scheduled daily exercise on free-running circadian period in syrian ham...
6440828 - Left ventricular pump function in effort angina. influence of nitroglycerin, propranolo...
Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Chest     Volume:  121     ISSN:  0012-3692     ISO Abbreviation:  Chest     Publication Date:  2002 Mar 
Date Detail:
Created Date:  2002-03-12     Completed Date:  2002-04-09     Revised Date:  2004-11-17    
Medline Journal Info:
Nlm Unique ID:  0231335     Medline TA:  Chest     Country:  United States    
Other Details:
Languages:  eng     Pagination:  697-701     Citation Subset:  AIM; IM    
Affiliation:
Department of Respiratory Medicine, Graduate School of Medicine, Osaka City University, Abenoku, Osaka, Japan. kanazawa-h@med.osaka-cu.ac.jp
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Exercise / physiology*
Genotype
Humans
Male
Middle Aged
Oxygen / metabolism*
Polymorphism, Genetic*
Pulmonary Disease, Chronic Obstructive / genetics*,  physiopathology
Renin / genetics*
Chemical
Reg. No./Substance:
7782-44-7/Oxygen; EC 3.4.23.15/Renin

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Measurement of short-term changes in dyspnea and disease-specific quality of life following an acute...
Next Document:  Use of a mucus clearance device enhances the bronchodilator response in patients with stable COPD.