Document Detail


Assessment of microcirculatory remodeling with intracoronary flow velocity and pressure measurements: validation with endomyocardial sampling in cardiac allografts.
MedLine Citation:
PMID:  19805652     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
BACKGROUND: Intracoronary physiology techniques have been validated extensively for the assessment of epicardial stenoses but not for the lone study of coronary microcirculation. We performed a comparison between 4 intracoronary physiological indices with the actual structural microcirculatory changes documented in transplanted hearts. METHODS AND RESULTS: In 17 cardiac allograft patients without coronary stenoses, ECG, intracoronary Doppler flow velocity, and aortic pressure were digitally recorded before and during maximal hyperemia with a dedicated system. Postprocessing of data yielded 4 indices of microcirculatory status: coronary flow velocity reserve (2.13+/-0.59), instantaneous hyperemic diastolic velocity pressure slope (2.33+/-1.25 cm x s x (-1)mm Hg(-1)), coronary resistance index (1.65+/-0.88 mm Hg x cm(-1) x s(-1)), and coronary resistance reserve (2.36+/-0.65). Quantitative morphometry was performed in endomyocardial biopsies during the same hospital intake; arteriolar obliteration (76.57+/-6.95%) and density (2.00+/-1.22 arterioles per 1 mm(2)) and capillary density (645+/-179 capillaries per 1 mm(2)) were measured. Univariate regression analysis between intracoronary measurements and histological findings revealed that instantaneous hyperemic diastolic velocity-pressure slope correlated with arteriolar obliteration (r=0.58, P=0.014) and capillary density (r=0.60, P=0.012). Statistical adjustment revealed an independent contribution of arteriolar obliteration (beta=0.61, P=0.0009) and capillary density (beta=-0.60, P=0.0008) to instantaneous hyperemic diastolic velocity-pressure slope values, resulting in an excellent predictive model (r=0.84, P=0.0002). Coronary resistance index correlated only with capillary density (r=0.70, P=0.019). Relative indices (coronary flow velocity reserve and coronary resistance reserve) did not correlate significantly with arteriolar obliteration, capillary density, or arteriolar density. CONCLUSIONS: Intracoronary indices derived from pressure and flow, particularly instantaneous hyperemic diastolic velocity-pressure slope, appear to be superior to coronary flow velocity reserve in detecting structural microcirculatory changes. Both arteriolar obliteration and capillary rarefaction seem to influence microcirculatory hemodynamics independently.
Authors:
Javier Escaned; Alex Flores; Pablo García-Pavía; Javier Segovia; Jesús Jimenez; Paloma Aragoncillo; Clara Salas; Fernando Alfonso; Rosana Hernández; Dominick J Angiolillo; Pilar Jiménez-Quevedo; Camino Bañuelos; Luis Alonso-Pulpón; Carlos Macaya
Publication Detail:
Type:  Comparative Study; Journal Article; Validation Studies     Date:  2009-10-05
Journal Detail:
Title:  Circulation     Volume:  120     ISSN:  1524-4539     ISO Abbreviation:  Circulation     Publication Date:  2009 Oct 
Date Detail:
Created Date:  2009-10-20     Completed Date:  2009-11-13     Revised Date:  2010-08-03    
Medline Journal Info:
Nlm Unique ID:  0147763     Medline TA:  Circulation     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1561-8     Citation Subset:  AIM; IM    
Affiliation:
Cardiovascular Institute, Hospital Clínico San Carlos, Madrid, Spain. escaned@secardiologia.es
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MeSH Terms
Descriptor/Qualifier:
Adult
Arterioles / pathology
Blood Flow Velocity*
Blood Pressure*
Capillaries / pathology
Coronary Circulation*
Diastole
Endocardium / pathology
Female
Heart Transplantation*
Humans
Hyperemia / physiopathology
Male
Microvessels / physiopathology*
Middle Aged
Myocardium / pathology
Predictive Value of Tests
Transplantation, Homologous
Vascular Resistance
Young Adult
Comments/Corrections
Comment In:
Circulation. 2010 Jul 27;122(4):e404; author reply e405   [PMID:  20660811 ]
Circulation. 2009 Oct 20;120(16):1555-7   [PMID:  19805646 ]

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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