Document Detail


Asiatic acid, a pentacyclic triterpene from Centella asiatica, is neuroprotective in a mouse model of focal cerebral ischemia.
MedLine Citation:
PMID:  19382233     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Asiatic acid, a triterpenoid derivative from Centella asiatica, has shown biological effects such as antioxidant, antiinflammatory, and protection against glutamate- or beta-amyloid-induced neurotoxicity. We investigated the neuroprotective effect of asiatic acid in a mouse model of permanent cerebral ischemia. Various doses of asiatic acid (30, 75, or 165 mg/kg) were administered orally at 1 hr pre- and 3, 10, and 20 hr postischemia, and infarct volume and behavioral deficits were evaluated at day 1 or 7 postischemia. IgG (blood-brain barrier integrity) and cytochrome c (apoptosis) immunostaining was carried out at 24 hr postischemia. The effect of asiatic acid on stress-induced cytochrome c release was examined in isolated mitochondrial fractions. Furthermore, its effects on cell viability and mitochondrial membrane potential were studied in HT-22 cells exposed to oxygen-glucose deprivation. Asiatic acid significantly reduced the infarct volume by 60% at day 1 and by 26% at day 7 postischemia and improved neurological outcome at 24 hr postischemia. Our studies also showed that the neuroprotective properties of asiatic acid might be mediated in part through decreased blood-brain barrier permeability and reduction in mitochondrial injury. The present study suggests that asiatic acid may be useful in the treatment of cerebral ischemia.
Authors:
Rajanikant G Krishnamurthy; Marie-Claude Senut; Daniel Zemke; Jiangyong Min; Mark B Frenkel; Eric J Greenberg; Seong-Woon Yu; Nick Ahn; John Goudreau; Mounzer Kassab; Kiran S Panickar; Arshad Majid
Related Documents :
11234913 - Determination of folate derivatives in rat tissues during folate deficiency.
6878453 - The maternal pheromone of the rat: testing some assumptions underlying a hypothesis.
11350053 - Overexpression of ucp-3 in skeletal muscle of mice results in increased expression of m...
Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Journal of neuroscience research     Volume:  87     ISSN:  1097-4547     ISO Abbreviation:  J. Neurosci. Res.     Publication Date:  2009 Aug 
Date Detail:
Created Date:  2009-07-07     Completed Date:  2009-09-21     Revised Date:  2011-01-12    
Medline Journal Info:
Nlm Unique ID:  7600111     Medline TA:  J Neurosci Res     Country:  United States    
Other Details:
Languages:  eng     Pagination:  2541-50     Citation Subset:  IM    
Affiliation:
Division of Cerebrovascular Diseases and Department of Neurology and Ophthalmology, Michigan State University, East Lansing, Michigan 48824, USA.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Brain / drug effects,  metabolism,  pathology
Brain Ischemia / drug therapy*
Cell Hypoxia
Cell Line
Cell Survival / drug effects
Cytochromes c / metabolism
Disease Models, Animal
Glucose / deficiency
Immunoglobulin G / metabolism
Infarction, Middle Cerebral Artery / drug therapy*
Male
Membrane Potential, Mitochondrial / drug effects
Mice
Mice, Inbred C57BL
Mitochondria / drug effects,  metabolism
Neuroprotective Agents / administration & dosage,  therapeutic use*
Pentacyclic Triterpenes
Severity of Illness Index
Time Factors
Treatment Outcome
Triterpenes / administration & dosage,  therapeutic use*
Grant Support
ID/Acronym/Agency:
P30 AG028717-02/AG/NIA NIH HHS
Chemical
Reg. No./Substance:
0/Immunoglobulin G; 0/Neuroprotective Agents; 0/Pentacyclic Triterpenes; 0/Triterpenes; 464-92-6/asiatic acid; 50-99-7/Glucose; 9007-43-6/Cytochromes c

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Comparative anatomy and evolution of the cardiac innervation in New World monkeys (Platyrrhini, e. G...
Next Document:  Functional and immunocytochemical characterization of D-serine transporters in cortical neuron and a...