Document Detail


Arterial stiffness: a brief review.
MedLine Citation:
PMID:  20802505     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Physical stiffening of the large arteries is the central paradigm of vascular aging. Indeed, stiffening in the larger central arterial system, such as the aortic tree, significantly contributes to cardiovascular diseases in older individuals and is positively associated with systolic hypertension, coronary artery disease, stroke, heart failure and atrial fibrillation, which are the leading causes of mortality in the developed countries and also in the developing world as estimated in 2010 by World Health Organizations. Thus, better, less invasive and more accurate measures of arterial stiffness have been developed, which prove useful as diagnostic indices, pathophysiological markers and predictive indicators of disease. This article presents a review of the structural determinants of vascular stiffening, its pathophysiologic determinants and its implications for vascular research and medicine. A critical discussion of new techniques for assessing vascular stiffness is also presented.
Authors:
Najeeb A Shirwany; Ming-hui Zou
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Review     Date:  2010-08-30
Journal Detail:
Title:  Acta pharmacologica Sinica     Volume:  31     ISSN:  1745-7254     ISO Abbreviation:  Acta Pharmacol. Sin.     Publication Date:  2010 Oct 
Date Detail:
Created Date:  2010-10-05     Completed Date:  2011-02-17     Revised Date:  2012-05-07    
Medline Journal Info:
Nlm Unique ID:  100956087     Medline TA:  Acta Pharmacol Sin     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1267-76     Citation Subset:  IM    
Affiliation:
Department of Medicine, University of Oklahoma Health Science Center, Oklahoma City, 73104, USA.
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MeSH Terms
Descriptor/Qualifier:
Aging / physiology
Animals
Arteries / metabolism,  physiopathology*
Cardiovascular Diseases / genetics,  metabolism,  physiopathology*
Diagnostic Techniques, Cardiovascular
Diet
Elasticity*
Endothelium, Vascular / physiopathology
Glucose / physiology
Humans
Insulin / physiology
Neurosecretory Systems / physiopathology
Risk Factors
Vascular Resistance*
Grant Support
ID/Acronym/Agency:
HL074399/HL/NHLBI NIH HHS; HL079584/HL/NHLBI NIH HHS; HL080499/HL/NHLBI NIH HHS; HL089920/HL/NHLBI NIH HHS; HL096032/HL/NHLBI NIH HHS; R01 HL074399-05/HL/NHLBI NIH HHS; R01 HL074399-06/HL/NHLBI NIH HHS; R01 HL074399-07/HL/NHLBI NIH HHS; R01 HL074399-08/HL/NHLBI NIH HHS; R01 HL079584-07/HL/NHLBI NIH HHS; R01 HL079584-08/HL/NHLBI NIH HHS; R01 HL079584-09/HL/NHLBI NIH HHS; R01 HL080499-04/HL/NHLBI NIH HHS; R01 HL080499-05/HL/NHLBI NIH HHS; R01 HL089920-03/HL/NHLBI NIH HHS; R01 HL089920-04/HL/NHLBI NIH HHS; R01 HL096032-02/HL/NHLBI NIH HHS; R01 HL096032-03/HL/NHLBI NIH HHS; R01 HL096032-04/HL/NHLBI NIH HHS; R01 HL105157-01/HL/NHLBI NIH HHS; R01 HL105157-02/HL/NHLBI NIH HHS; R01 HL105157-03/HL/NHLBI NIH HHS; R01 HL110488-02/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
0/Insulin; 50-99-7/Glucose
Comments/Corrections

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