Document Detail


Arsenic trioxide-induced apoptosis in H9c2 cardiomyocytes: implications in cardiotoxicity.
MedLine Citation:
PMID:  18346055     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Arsenic trioxide (As(2)O(3)) achieved dramatic remissions in patients with acute promyelocytic leukaemia. Clinical reports have shown that treatment was associated with cardiotoxicity. We investigated the toxic mechanisms of As(2)O(3) in H9c2 cardiomyocytes. Clinically relevant concentrations of As(2)O(3) (2-10 microM) reduced the viability of H9c2 cells in a concentration-dependent manner. The decreased cell viability was because As(2)O(3) induced cell apoptosis (cell shrinkage, nuclear alterations and caspase-3 activation), or even necrosis at higher concentrations. Inhibition of caspase-3 with a specific inhibitor, Ac-DEVD-CHO, suppressed apoptosis induced by As(2)O(3). In addition, reactive oxygen species formation and cellular Ca(2+) overload were observed in H9c2 cells exposed to As(2)O(3), which was partly inhibited by vitamin E and verapamil. These results suggest that As(2)O(3)-induced cardiotoxicity is mediated, at least in part, by activation of caspase-3 pathway, which may be triggered by reactive oxygen species formation and intracellular Ca(2+) overload.
Authors:
Xiaoyan Zhao; Tieming Feng; He Chen; Hongli Shan; Yong Zhang; Yanjie Lu; Baofeng Yang
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2008-03-12
Journal Detail:
Title:  Basic & clinical pharmacology & toxicology     Volume:  102     ISSN:  1742-7843     ISO Abbreviation:  Basic Clin. Pharmacol. Toxicol.     Publication Date:  2008 May 
Date Detail:
Created Date:  2008-04-17     Completed Date:  2008-07-17     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101208422     Medline TA:  Basic Clin Pharmacol Toxicol     Country:  Denmark    
Other Details:
Languages:  eng     Pagination:  419-25     Citation Subset:  IM    
Affiliation:
Department of Pharmacology, Harbin Medical University, Harbin, China.
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MeSH Terms
Descriptor/Qualifier:
Animals
Antineoplastic Agents / adverse effects*
Antioxidants / pharmacology
Apoptosis / drug effects*
Arsenicals / adverse effects*
Caspase 3 / metabolism
Cell Line
Cell Survival / drug effects
Cysteine Proteinase Inhibitors / pharmacology
Dose-Response Relationship, Drug
Enzyme Activation
Myocytes, Cardiac / cytology,  drug effects*,  metabolism
Oligopeptides / pharmacology
Oxidative Stress / drug effects
Oxides / adverse effects*
Rats
Reactive Oxygen Species / metabolism
Verapamil / pharmacology
Vitamin E / pharmacology
Chemical
Reg. No./Substance:
0/Antineoplastic Agents; 0/Antioxidants; 0/Arsenicals; 0/Cysteine Proteinase Inhibitors; 0/Oligopeptides; 0/Oxides; 0/Reactive Oxygen Species; 0/acetyl-aspartyl-glutamyl-valyl-aspartal; 1327-53-3/arsenic trioxide; 1406-18-4/Vitamin E; 52-53-9/Verapamil; EC 3.4.22.-/Caspase 3

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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