Document Detail


Apoptosis induced by inhibition of contact with extracellular matrix in mouse collecting duct cells.
MedLine Citation:
PMID:  10575296     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Cell-matrix interactions have major effects upon phenotypic features such as gene regulation, cytoskeletal structure, differentiation and aspects of cell growth control. Detachment from the matrix epithelial cells induces programmed cell death, and this cell detachment induced apoptosis has been referred to as 'anoikis'. This study was undertaken to determine whether apoptosis is induced by inhibition of contact with extracellular matrix (ECM) in collecting duct cells and to investigate the signaling mechanisms of the process. Upon detachment from ECM, mouse inner medullary collecting duct cells (mIMCD-3) and mouse outer cortical collecting duct cells (M-1), which were derived from an SV40 transgenic mouse, entered into programmed cell death. Forced suspension of mIMCD-3 or M-1 cells did not affect the expression of Bcl-2-related proteins and did not activate c-Jun NH(2)-terminal kinase. Detachment of cells from ECM activated p38 mitogen-activated protein kinase (p38), but its inhibition with SB203580 did not protect cells from anoikis. Detachment of cells from matrix inhibited NF-kappaB activity, and the inhibition of NF-kappaB activity by overexpression of nonphosphorylatable I-kappaB increased detachment-induced apoptotic cell death in M-1 cells. Forced suspension of M-1 cells still activated p53 activity. Caspase-8 was activated during anoikis, but the time course of its activation was in accordance with DNA fragmentation. These results indicate that detachment from ECM induces apoptosis in the kidney collecting duct cells. Changes in expression levels of Bcl-2-related proteins or activation of JNK/p38 kinase are not critical for anoikis. Decrease in NF-kappaB activity and activation of p53 induced by inhibition of interaction with ECM play roles in anoikis in SV-40-transformed collecting duct cells. Caspase-8 is activated during detachment-induced apoptosis, the mechanisms of which are independent of activation of cell death receptors.
Authors:
M Y Park; R H Lee; S H Lee; J S Jung
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Nephron     Volume:  83     ISSN:  0028-2766     ISO Abbreviation:  Nephron     Publication Date:  1999  
Date Detail:
Created Date:  2000-02-04     Completed Date:  2000-02-04     Revised Date:  2010-01-15    
Medline Journal Info:
Nlm Unique ID:  0331777     Medline TA:  Nephron     Country:  SWITZERLAND    
Other Details:
Languages:  eng     Pagination:  341-51     Citation Subset:  IM    
Copyright Information:
Copyright 1999 S. Karger AG, Basel
Affiliation:
Department of Physiology, College of Medicine, Pusan National University, Pusan, Korea.
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MeSH Terms
Descriptor/Qualifier:
Animals
Apoptosis / physiology*
Base Sequence
Caspase 8
Caspase 9
Caspases / metabolism
Cell Adhesion / physiology*
Cell Line
DNA Primers / genetics
Epithelial Cells / cytology,  physiology
Extracellular Matrix / physiology*
JNK Mitogen-Activated Protein Kinases
Kidney Tubules, Collecting / cytology*,  physiology*
Mice
Mitogen-Activated Protein Kinases / metabolism
Proto-Oncogene Proteins / genetics,  metabolism
Proto-Oncogene Proteins c-bcl-2 / genetics,  metabolism
RNA, Messenger / genetics,  metabolism
bcl-2-Associated X Protein
bcl-X Protein
p38 Mitogen-Activated Protein Kinases
Chemical
Reg. No./Substance:
0/Bcl2l1 protein, mouse; 0/DNA Primers; 0/Proto-Oncogene Proteins; 0/Proto-Oncogene Proteins c-bcl-2; 0/RNA, Messenger; 0/bcl-2-Associated X Protein; 0/bcl-X Protein; EC 2.7.11.24/JNK Mitogen-Activated Protein Kinases; EC 2.7.11.24/Mitogen-Activated Protein Kinases; EC 2.7.11.24/p38 Mitogen-Activated Protein Kinases; EC 3.4.22.-/Casp8 protein, mouse; EC 3.4.22.-/Casp9 protein, mouse; EC 3.4.22.-/Caspase 8; EC 3.4.22.-/Caspase 9; EC 3.4.22.-/Caspases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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