Document Detail


Apoptosis in the absence of poly-(ADP-ribose) polymerase.
MedLine Citation:
PMID:  9144569     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Cleavage of poly-(ADP-ribose) polymerase is a process occurring early during the execution phase of apoptosis. Although in many experimental systems PARP cleavage indicates a point of no return, the significance of this proteolytic step for apoptosis remains unclear. Here we compare the susceptibility of cells from wild-type mice and PARP-/- mice to several inducers of apoptosis. Neither the susceptibility of hepatocytes towards CD95 or TNF-mediated apoptosis nor the activation of PARP-cleaving caspases was modified in PARP-/- liver cells. Thymocytes with either genotype exhibited similar sensitivity to treatments with ceramide, dexamethasone, or etoposide. The sensitivity of primary neurons towards apoptosis induced by staurosporine, colchicine, potassium withdrawal, peroxynitrite, or the neurotoxin MPP+ was also unaltered. These data suggest that neither activation nor cleavage of PARP has a causal role in apoptotic cell death of primary, non-transformed cells.
Authors:
M Leist; B Single; G Künstle; C Volbracht; H Hentze; P Nicotera
Related Documents :
11070499 - Antioxidants j811 and 17beta-estradiol protect cerebellar granule cells from methylmerc...
19147789 - Parp-14, a member of the b aggressive lymphoma family, transduces survival signals in p...
22467879 - Arsenic promotes ubiquitinylation and lysosomal degradation of cystic fibrosis transmem...
12657159 - Prostaglandin f2alpha- and fas-activating antibody-induced regression of the corpus lut...
10601269 - Survival and proliferation of cells expressing caspase-uncleavable poly(adp-ribose) pol...
19900759 - Tuning of apoptosis-mediator gene transcription in hepg2 human hepatoma cells through a...
12952289 - Autologous adjuvant linked fibroblasts induce anti-glioma immunity: implications for de...
6300969 - Production of cyclooxygenase products and superoxide anion by macrophages in response t...
12515809 - Mycobacteria target dc-sign to suppress dendritic cell function.
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Biochemical and biophysical research communications     Volume:  233     ISSN:  0006-291X     ISO Abbreviation:  Biochem. Biophys. Res. Commun.     Publication Date:  1997 Apr 
Date Detail:
Created Date:  1997-06-05     Completed Date:  1997-06-05     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  0372516     Medline TA:  Biochem Biophys Res Commun     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  518-22     Citation Subset:  IM    
Affiliation:
Department of Molecular Toxicology, University of Konstanz, Germany.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Apoptosis*
Cells, Cultured
Electrophoresis, Agar Gel
L-Lactate Dehydrogenase / metabolism
Liver / metabolism
Mice
Microscopy, Fluorescence
Poly(ADP-ribose) Polymerases / metabolism*
Tumor Necrosis Factor-alpha / metabolism
Chemical
Reg. No./Substance:
0/Tumor Necrosis Factor-alpha; EC 1.1.1.27/L-Lactate Dehydrogenase; EC 2.4.2.30/Poly(ADP-ribose) Polymerases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  A novel Mn++-dependent ribonuclease that functions in U16 SnoRNA processing in X. laevis.
Next Document:  Expression of human inducible nitric oxide synthase is regulated by both promoter and 3'-regions.