Document Detail


Apoptosis and eosinophils. Regulation and clinical relevance.
MedLine Citation:
PMID:  18454208     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Apoptosis, or program cell death, is a process of fundamental biological importance, and eosinophil apoptosis is believed to be the primary mechanism for removing eosinophils from the lung followed by their recognition and phagocytosis by macrophages or resident bronchial epithelial cells. There is, therefore, an increased interest in the fundamental role of the signals and intracellular signaling molecules that initiate and control apoptosis in human eosinophils though much remains to be established. This article reviews briefly the cross talks between apoptosis and eosinophils and summarizes the recent developments in this field.
Authors:
Faris Q Alenzi
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Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Saudi medical journal     Volume:  29     ISSN:  0379-5284     ISO Abbreviation:  Saudi Med J     Publication Date:  2008 May 
Date Detail:
Created Date:  2008-05-05     Completed Date:  2008-09-04     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  7909441     Medline TA:  Saudi Med J     Country:  Saudi Arabia    
Other Details:
Languages:  eng     Pagination:  643-56     Citation Subset:  IM    
Affiliation:
Department of Clinical Laboratory Sciences, College of Applied Medical Sciences, King Saud University, PO Box 422, Al-Kharaj, Kingdom of Saudi Arabia. falenzi@ksu.edu.sa
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MeSH Terms
Descriptor/Qualifier:
Animals
Apoptosis / physiology*
Asthma / metabolism,  physiopathology
Cell Adhesion Molecules / metabolism
Cell Aging
Chemokines / metabolism
Cytokines / metabolism
Eosinophils / cytology,  metabolism,  physiology*
Humans
Hypersensitivity / metabolism,  physiopathology
Lung / cytology,  metabolism,  physiology,  physiopathology
Phagocytosis
Signal Transduction / physiology
Chemical
Reg. No./Substance:
0/Cell Adhesion Molecules; 0/Chemokines; 0/Cytokines

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