Document Detail


Apomorphine-haloperidol interactions: different types of antagonism in cortical and subcortical brain regions.
MedLine Citation:
PMID:  7470869     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Apomorphine, a dopamine (DA) receptor stimulant induced a dose-dependent decrease in the content of the dopamine metabolites 3,4-dihydroxyphenylacetic acid (DOPAC) and homovanillic acid (HVA) in the olfactory tubercle and striatum of rats, but it was ineffective in the frontal cortex. Apomorphine also antagonized the haloperidol-induced accumulation of DOPAC and HAV in the olfactory tubercle and striatum, whereas in the frontal cortex it antagonized the effects of haloperidol on DOPAC but not on HVA. The total (free and conjugated) content of DA metabolites was measured in these experiments.
Authors:
N G Bacopoulos; R H Roth
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Brain research     Volume:  205     ISSN:  0006-8993     ISO Abbreviation:  Brain Res.     Publication Date:  1981 Feb 
Date Detail:
Created Date:  1981-05-21     Completed Date:  1981-05-21     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  0045503     Medline TA:  Brain Res     Country:  NETHERLANDS    
Other Details:
Languages:  eng     Pagination:  313-9     Citation Subset:  IM    
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MeSH Terms
Descriptor/Qualifier:
3,4-Dihydroxyphenylacetic Acid / metabolism
Animals
Apomorphine / pharmacology*
Caudate Nucleus / drug effects
Corpus Striatum / drug effects*
Dopamine / metabolism
Dose-Response Relationship, Drug
Frontal Lobe / drug effects*
Haloperidol / pharmacology*
Homovanillic Acid / metabolism
Male
Olfactory Bulb / drug effects*
Rats
Receptors, Dopamine / drug effects
Grant Support
ID/Acronym/Agency:
MH-14092/MH/NIMH NIH HHS
Chemical
Reg. No./Substance:
0/Receptors, Dopamine; 102-32-9/3,4-Dihydroxyphenylacetic Acid; 306-08-1/Homovanillic Acid; 52-86-8/Haloperidol; 58-00-4/Apomorphine

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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