Document Detail


Antioxidant enzyme inhibitors enhance singlet oxygen-induced cell death in HL-60 cells.
MedLine Citation:
PMID:  17018401     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Singlet oxygen is a highly reactive form of molecular oxygen that may harm living systems by oxidizing critical cellular macromolecules and it also promotes deleterious processes such as cell death. The protective role of antioxidant enzymes against singlet oxygen-induced oxidative damage in HL-60 cells was investigated in control and cells pre-treated with diethyldithiocarbamic acid, aminotriazole and oxlalomalate, specific inhibitors of superoxide dismutase, catalase and NADP+-dependent isocitrate dehydrogenase, respectively. Upon exposure to rose bengal (20 microM)/light (15 min), which generates singlet oxygen, to HL-60 cells, the viability was lower and the lipid peroxidation and oxidative DNA damage were higher in inhibitor-treated cells as compared to control cells. We also observed the significant increase in the endogenous production of reactive oxygen species as well as the significant decrease in the intracellular GSH level in inhibitor-treated HL-60 cells exposed to singlet oxygen. Upon exposure to rose bengal (3 microM)/light (15 min), which induced apoptotic cell death, a clear inverse relationship was observed between the control and inhibitor-treated HL-60 cells in their susceptibility to apoptosis. These results suggest that antioxidant enzymes play an important role in cellular defense against singlet oxygen-induced cell death including necrosis and apoptosis.
Authors:
Sun Yee Kim; Su Min Lee; Jeen-Woo Park
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Free radical research     Volume:  40     ISSN:  1071-5762     ISO Abbreviation:  Free Radic. Res.     Publication Date:  2006 Nov 
Date Detail:
Created Date:  2006-10-04     Completed Date:  2007-02-07     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9423872     Medline TA:  Free Radic Res     Country:  England    
Other Details:
Languages:  eng     Pagination:  1190-7     Citation Subset:  IM    
Affiliation:
School of Life Sciences and Biotechnology, College of Natural Sciences, Kyungpook National University, Taegu, 702-701, South Korea.
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MeSH Terms
Descriptor/Qualifier:
Antioxidants / metabolism,  pharmacology*
Apoptosis
Cell Death
Cell Separation
DNA Damage
Enzyme Inhibitors / pharmacology*
Glutathione / metabolism
HL-60 Cells
Humans
Lipid Peroxidation
Necrosis
Oxidation-Reduction
Oxidative Stress
Oxygen / metabolism*
Singlet Oxygen / metabolism
Chemical
Reg. No./Substance:
0/Antioxidants; 0/Enzyme Inhibitors; 17778-80-2/Singlet Oxygen; 70-18-8/Glutathione; 7782-44-7/Oxygen

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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