Document Detail


Antiinflammatory therapy in myocarditis.
MedLine Citation:
PMID:  12826819     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Acute viral myocarditis progresses through several stages, including acute viral infection, autoimmunity, and dilated cardiomyopathy. Recent studies have identified an important role for the myocardial inflammatory response during the initial phases of acute viral myocarditis. In this review, we discuss new insights into the innate host response to viral myocarditis and the various therapeutic strategies that have been attempted in managing the deleterious consequences of the myocardial inflammatory response in acute viral myocarditis.
Authors:
Jesus Vallejo; Douglas L Mann
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Publication Detail:
Type:  Journal Article; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.; Review    
Journal Detail:
Title:  Current opinion in cardiology     Volume:  18     ISSN:  0268-4705     ISO Abbreviation:  Curr. Opin. Cardiol.     Publication Date:  2003 May 
Date Detail:
Created Date:  2003-06-26     Completed Date:  2003-12-04     Revised Date:  2008-11-21    
Medline Journal Info:
Nlm Unique ID:  8608087     Medline TA:  Curr Opin Cardiol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  189-93     Citation Subset:  IM    
Affiliation:
Winters Center for Heart Failure Research, Cardiology Section, Medical Care Line, Veterans Affairs Medical Center, and Department of Medicine, Baylor College of Medicine, Houston, TX 77030, USA.
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MeSH Terms
Descriptor/Qualifier:
Acute Disease
Adjuvants, Immunologic / therapeutic use
Humans
Immunity, Innate
Immunosuppressive Agents / therapeutic use
Immunotherapy / methods*
Membrane Glycoproteins / metabolism
Myocarditis / immunology*,  therapy*,  virology
Receptors, Cell Surface / metabolism
Toll-Like Receptors
Grant Support
ID/Acronym/Agency:
HL_4225_10/10/HL/NHLBI NIH HHS; P50 HL_O6H/HL/NHLBI NIH HHS; R01 HL58081_01/HL/NHLBI NIH HHS; R01 HL61543_01/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
0/Adjuvants, Immunologic; 0/Immunosuppressive Agents; 0/Membrane Glycoproteins; 0/Receptors, Cell Surface; 0/Toll-Like Receptors

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