Document Detail


Anti-angiogenic effect of high-dose resveratrol in a swine model of metabolic syndrome.
MedLine Citation:
PMID:  20570307     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
BACKGROUND: Resveratrol has been reported to induce angiogenesis in ischemic tissue. We hypothesized that high-dose resveratrol would improve native angiogenesis in a swine model of metabolic syndrome and chronic myocardial ischemia.
METHODS: Yorkshire swine were fed a normal diet (Control, n = 7), hypercholesterolemic diet (HCD, n = 7), or hypercholesterolemic diet with supplemental resveratrol (100 mg/kg/day orally, HCD-R; n = 7) beginning 1 month prior to surgery. Chronic ischemia was created by placing an ameroid constrictor on the left circumflex coronary artery. After 7 weeks, swine underwent functional MRI, coronary angiography, and serum and heart tissue harvest for analysis.
RESULTS: HCD-R animals had lower body mass index (P < .001), total cholesterol (P < .001), low-density lipoprotein (LDL; P < .001), blood glucose levels (P < .001), and systolic blood pressure (P = .03) than HCD animals. There was no difference in regional myocardial function at 7 weeks (P = .25). Coronary angiograms revealed no difference in Rentrop collateral scores (P = .68). Staining for platelet endothelial cell adhesion molecule-1 demonstrated higher capillary density in the Control group (versus HCD and HCD-R; P = .02). Immunoblotting demonstrated decreased expression of the pro-angiogenic protein vascular endothelial (VE)-cadherin (P = .002) and an increase in anti-angiogenic proteins angiostatin (P = .001) and thrombospondin (P = .02) in the HCD and HCD-R groups. Matrix metalloprotease 2 (MMP 2; P = .47) and MMP 9 (P = .12) were not different among groups.
CONCLUSION: Supplemental resveratrol positively modified cardiovascular risk factors including body mass index, cholesterol, glucose tolerance, and systolic blood pressure. However, it did not increase native collateral formation in the ischemic myocardium. This may be a result of increased angiostatin and thrombospondin leading to decreased expression of VE-cadherin and other pro-angiogenic factors.
Authors:
Michael P Robich; Louis M Chu; Mirnal Chaudray; Reza Nezafat; Yuchi Han; Richard T Clements; Roger J Laham; Warren J Manning; Michael A Coady; Frank W Sellke
Related Documents :
11311217 - Correlation between total homocysteine, low-density lipoprotein cholesterol and high-de...
3134897 - Dyslipoproteinemia in pediatric systemic lupus erythematosus.
6440737 - Effect of subdermal levonorgestrel contraceptive implants, norplant, on serum lipids.
15905737 - Lipodystrophy and dyslipidemia among patients taking first-line, world health organizat...
14974547 - Effect of forage quality on digestion and performance responses of cattle to supplement...
11311217 - Correlation between total homocysteine, low-density lipoprotein cholesterol and high-de...
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2010-06-08
Journal Detail:
Title:  Surgery     Volume:  148     ISSN:  1532-7361     ISO Abbreviation:  Surgery     Publication Date:  2010 Aug 
Date Detail:
Created Date:  2010-07-16     Completed Date:  2010-08-06     Revised Date:  2011-08-03    
Medline Journal Info:
Nlm Unique ID:  0417347     Medline TA:  Surgery     Country:  United States    
Other Details:
Languages:  eng     Pagination:  453-62     Citation Subset:  AIM; IM    
Copyright Information:
Copyright 2010 Mosby, Inc. All rights reserved.
Affiliation:
Department of Surgery, Division of Cardiothoracic Surgery, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA, USA.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Angiogenesis Inhibitors / administration & dosage
Angiostatins / metabolism
Animals
Antigens, CD / metabolism
Cadherins / metabolism
Cholesterol, Dietary / administration & dosage
Collateral Circulation / drug effects
Coronary Angiography
Disease Models, Animal
Magnetic Resonance Imaging
Male
Metabolic Syndrome X / drug therapy*,  metabolism,  pathology
Myocardial Ischemia / drug therapy,  metabolism,  pathology
Neovascularization, Physiologic / drug effects*
Stilbenes / administration & dosage*
Swine
Swine, Miniature
Grant Support
ID/Acronym/Agency:
R01 HL046716-18A1/HL/NHLBI NIH HHS; R01 HL069024-08/HL/NHLBI NIH HHS; R01 HL069024-09/HL/NHLBI NIH HHS; R01 HL085647-03/HL/NHLBI NIH HHS; R01HL46716/HL/NHLBI NIH HHS; R01HL69024/HL/NHLBI NIH HHS; R01HL85647/HL/NHLBI NIH HHS; T32 HL076130-05/HL/NHLBI NIH HHS; T32-HL0074/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
0/Angiogenesis Inhibitors; 0/Antigens, CD; 0/Cadherins; 0/Cholesterol, Dietary; 0/Stilbenes; 0/cadherin 5; 501-36-0/resveratrol; 86090-08-6/Angiostatins
Comments/Corrections

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  New prognostic factor influencing long-term survival of patients with advanced gallbladder carcinoma...
Next Document:  Estimate of the attenuation coefficient using a clinical array transducer for the detection of cervi...