Document Detail


Antenatal betamethasone alters vascular reactivity in adult female ovine cerebral arteries.
MedLine Citation:
PMID:  20581740     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Although the use of antenatal glucocorticoids has resulted in decreased neonatal morbidity/mortality, recent animal studies have raised concerns regarding adverse effects of these medications on postnatal cardiovascular function. We hypothesized that antenatal betamethasone (Beta) exposure alters cerebral vascular reactivity in adult female sheep. We observed that K-induced constriction was comparable in middle cerebral artery (MCA) from Beta-exposed animals and age-matched controls. Pressure-induced constriction was significantly attenuated in MCA from Beta-exposed compared with control sheep. Inhibition of NOS significantly augmented pressure-induced constriction in MCA from both Beta-exposed and control sheep, whereas cyclooxygenase (COX) inhibition augmented pressure-induced constriction only in MCA from Beta-exposed sheep. Furthermore, NOS and COX inhibition significantly attenuated bradykinin (BK)-induced dilation in MCA from both Beta-exposed and control sheep. However, there seemed to be a greater contribution of both NOS and COX to BK-induced dilation in Beta-exposed compared with control MCA. Our findings demonstrate that fetal exposure to a clinically relevant course of Beta alters cerebral vascular tone and reactivity in adult female sheep.
Authors:
Delrae M Eckman; Brady A Kerr; Mamta Fuloria; Steve A Simandle; Suzanne E Watt; James C Rose; Jorge P Figueroa
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural    
Journal Detail:
Title:  Pediatric research     Volume:  68     ISSN:  1530-0447     ISO Abbreviation:  Pediatr. Res.     Publication Date:  2010 Oct 
Date Detail:
Created Date:  2010-09-16     Completed Date:  2011-01-11     Revised Date:  2011-10-19    
Medline Journal Info:
Nlm Unique ID:  0100714     Medline TA:  Pediatr Res     Country:  United States    
Other Details:
Languages:  eng     Pagination:  344-8     Citation Subset:  IM    
Affiliation:
Department of Pediatrics, Wake Forest University School of Medicine, Winston-Salem, North Carolina 27157, USA. deckman@wfubmc.edu
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MeSH Terms
Descriptor/Qualifier:
Age Factors
Animals
Betamethasone / administration & dosage*,  toxicity
Blood Pressure
Cyclooxygenase Inhibitors / pharmacology
Dose-Response Relationship, Drug
Enzyme Inhibitors / pharmacology
Female
Glucocorticoids / administration & dosage*,  toxicity
Middle Cerebral Artery / drug effects*,  enzymology
Nitric Oxide Synthase / antagonists & inhibitors,  metabolism
Pregnancy
Prenatal Exposure Delayed Effects*
Sheep
Vasoconstriction / drug effects*
Vasodilation / drug effects*
Vasodilator Agents / pharmacology
Grant Support
ID/Acronym/Agency:
HL068728/HL/NHLBI NIH HHS; P01 HD047584/HD/NICHD NIH HHS; P01 HD047584-02/HD/NICHD NIH HHS; P01 HD047584-03/HD/NICHD NIH HHS; R01 HD017644-25/HD/NICHD NIH HHS; R01 HL068728-07/HL/NHLBI NIH HHS; R01 HL068728-08/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
0/Cyclooxygenase Inhibitors; 0/Enzyme Inhibitors; 0/Glucocorticoids; 0/Vasodilator Agents; 378-44-9/Betamethasone; EC 1.14.13.39/Nitric Oxide Synthase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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