Document Detail


Angiotensin II and cell cycle regulation.
MedLine Citation:
PMID:  14967829     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Angiotensin II has emerged as an important growth factor for vascular, cardiac, and renal cells. Depending on the specific cell type and presence of other growth factors, angiotensin II induces proliferation (replication of DNA with subsequent successful division of cells), hypertrophy (increase in cell size, cell protein, and mRNA content without DNA replication), apoptosis (programmed cell death), or differentiation. Such angiotensin II-mediated modulation of growth process may underlie various pathophysiological processes such as atherosclerosis, vascular and cardiac remodeling, and progression of chronic renal disease. Clearly, angiotensin II-induced proliferation requires complete cell progression through the various steps of the cell cycle. In contrast, cells undergoing angiotensin II-mediated hypertrophy are arrested in the G1-phase. Upregulation of cell cycle-dependent kinase inhibitors (eg, p27Kip1) plays an important role in this process. Although accumulating evidence suggests that apoptosis is cell cycle-dependent, only few data are currently available concerning the interaction of angiotensin II with the cell cycle machinery in apoptosis. We review the various angiotensin II-mediated growth processes and their relationship to events governing cell cycle regulation.
Authors:
Gunter Wolf; Ulrich O Wenzel
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Review     Date:  2004-02-16
Journal Detail:
Title:  Hypertension     Volume:  43     ISSN:  1524-4563     ISO Abbreviation:  Hypertension     Publication Date:  2004 Apr 
Date Detail:
Created Date:  2004-03-26     Completed Date:  2004-08-10     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  7906255     Medline TA:  Hypertension     Country:  United States    
Other Details:
Languages:  eng     Pagination:  693-8     Citation Subset:  IM    
Affiliation:
Department of Medicine, Division of Nephrology and Osteology, University of Hamburg, University Hospital, Germany. Wolf@UKE.uni-hamburg.de
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MeSH Terms
Descriptor/Qualifier:
Angiotensin II / physiology*
Animals
Apoptosis / physiology
Blood Vessels / cytology,  growth & development,  metabolism
Cardiovascular Diseases / metabolism,  pathology
Cell Cycle / physiology*
Cell Cycle Proteins / physiology
Cell Differentiation / physiology
Cell Division / physiology
Cell Size
Endothelins / physiology
G1 Phase
Heart / growth & development,  physiology
Humans
Hypertrophy
Kidney / cytology,  growth & development,  metabolism
Kidney Diseases / metabolism,  pathology
Myocardium / cytology,  metabolism
Chemical
Reg. No./Substance:
0/Cell Cycle Proteins; 0/Endothelins; 11128-99-7/Angiotensin II

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