Document Detail

Angioedema: Differential diagnosis and treatment.
MedLine Citation:
PMID:  22195755     Owner:  NLM     Status:  In-Data-Review    
Conditions causing angioedema can be loosely classified as those with underlying C1-inhibitor deficiency or dysfunction and those that do not. Determining the root cause of symptoms and ruling out conditions that masquerade as angioedema help clinicians plan appropriate management. The various etiologies of angioedema have overlapping symptoms that can complicate the diagnosis. An awareness of features unique to a specific cause of angioedema will aid in the differential diagnosis. A thorough history may reveal a family history of episodic swelling, bouts of swelling associated with medication use, or swelling associated with certain foods or environmental allergens. Similarly, a history of symptom onset later in life is more common in acquired angioedema. The presence of urticaria suggests an allergic component. Treatment strategies have been devised for all forms of angioedema, although specific therapeutic targets may be unknown. Several medications directed at the underlying cause of symptoms in hereditary angioedema have been recently approved for use in the United States. Clinical symptoms, differential diagnosis, and management strategies for angioedema are reviewed in this article.
Paula J Busse
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Allergy and asthma proceedings : the official journal of regional and state allergy societies     Volume:  32 Suppl 1     ISSN:  1539-6304     ISO Abbreviation:  Allergy Asthma Proc     Publication Date:    2011 Sep-Oct
Date Detail:
Created Date:  2011-12-26     Completed Date:  -     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9603640     Medline TA:  Allergy Asthma Proc     Country:  United States    
Other Details:
Languages:  eng     Pagination:  3-11     Citation Subset:  IM    
Division of Clinical Immunology, Department of Medicine, Mount Sinai School of Medicine, New York, USA.
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