Document Detail


Analysis of neuronal nitric oxide synthase expression and increasing astrogliosis in the brain of senescence-accelerated-prone 8 mice.
MedLine Citation:
PMID:  20707635     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The senescence-accelerated mouse (SAM) is an autogenic senile murine model characterized by early cognitive impairment and age-related deterioration of learning and memory. The present study investigated the alternations of neuronal nitric oxide synthase (nNOS) expression in frontal cortex and hippocampus in the aging process of SAM-prone 8 (SAMP8) and SAM-resistant 1 (SAMR1) mice. The results demonstrated that the expression of nNOS was upregulated in the frontal cortex, but downregulated in the hippocampus in SAMP8. Further, age-related increases of astrogliosis were seen in the cortex and hippocampi of aged SAMP8 and SAMR1, as revealed by the expression of the astrocyte specific marker, glial fibrillary acidic protein (GFAP). Indeed, astrogliosis in aged SAMP8 was significantly greater than that of aged SAMR1. Our results suggest the possibility of a correlation between the downregulation of nitric oxide (NO) in the hippocampus and reported learning and memory deficits in SAMP8. However, the toxic effects of NO and age-related increases of astrogliosis, may have contributed to abnormal alterations in metabolism and neurochemical mechanisms in aged SAMP8.
Authors:
Shu Han; John A Rudd; Zhi Ying Hu; Lihong Zhang; David T Yew; Marong Fang
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  The International journal of neuroscience     Volume:  120     ISSN:  1563-5279     ISO Abbreviation:  Int. J. Neurosci.     Publication Date:  2010 Sep 
Date Detail:
Created Date:  2010-08-16     Completed Date:  2010-12-23     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0270707     Medline TA:  Int J Neurosci     Country:  England    
Other Details:
Languages:  eng     Pagination:  602-8     Citation Subset:  IM    
Affiliation:
Institute of Anatomy and Cell Biology, Medical College, Zhejiang University, China.
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MeSH Terms
Descriptor/Qualifier:
Aging / genetics,  metabolism*
Animals
Brain / metabolism*
Cerebral Cortex / metabolism
Glial Fibrillary Acidic Protein / metabolism
Gliosis / metabolism*
Hippocampus / metabolism
Male
Memory
Mice
Mice, Mutant Strains
Nitric Oxide / metabolism
Nitric Oxide Synthase Type I / biosynthesis*,  genetics
Chemical
Reg. No./Substance:
0/Glial Fibrillary Acidic Protein; 10102-43-9/Nitric Oxide; EC 1.14.13.39/Nitric Oxide Synthase Type I

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