Document Detail


Analysis of the human KCNH2(HERG) gene: identification and characterization of a novel mutation Y667X associated with long QT syndrome and a non-pathological 9 bp insertion.
MedLine Citation:
PMID:  10790218     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Long QT (LQT) syndrome is a potentially life-threatening disorder, characterized by a distinct cardiac arrhythmia known as torsades de pointes. Mutations within a number of genes linked to the familial form, including that coding for a cardiac potassium channel called KCNH2 (HERG), have been described based on the characterized genomic organization. A standardized method was developed to screen the entire gene for gene variants. We report a single base pair substitution, introducing a premature STOP codon at codon 667 of the gene in a healthy individual with an extended QTc interval (460 msec). In vitro expression of the codon Y667X variant in Xenopus oocyte suggests that the autosomal dominant variant does not function in a dominant/negative manner and cannot co-assemble to form a channel, resulting in a reduction of the KCNH2 current, and an extension of the QT interval. This indicates that pathogenic LQT gene variants exist in the apparently normal population, the prognosis and clinical consequences of which remain to be determined. The assays described should facilitate future studies into this area.
Authors:
A Paulussen; P Yang; M Pangalos; P Verhasselt; R Marrannes; C Verfaille; I Vandenberk; R Crabbe; F Konings; W Luyten; M Armstrong
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Human mutation     Volume:  15     ISSN:  1098-1004     ISO Abbreviation:  Hum. Mutat.     Publication Date:  2000 May 
Date Detail:
Created Date:  2000-09-21     Completed Date:  2000-09-21     Revised Date:  2008-10-28    
Medline Journal Info:
Nlm Unique ID:  9215429     Medline TA:  Hum Mutat     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  483     Citation Subset:  IM    
Copyright Information:
Copyright 2000 Wiley-Liss, Inc.
Affiliation:
Department of Pharmacogenomics, Janssen Research Foundation, Janssen Pharmaceutica, Belgium.
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MeSH Terms
Descriptor/Qualifier:
Animals
Cation Transport Proteins*
Codon / genetics
DNA-Binding Proteins*
Electrophysiology
Ether-A-Go-Go Potassium Channels
Humans
Long QT Syndrome / genetics*
Mutagenesis, Insertional / genetics*
Patch-Clamp Techniques
Potassium Channels / genetics*,  physiology
Potassium Channels, Voltage-Gated*
Trans-Activators*
Xenopus laevis / genetics
Chemical
Reg. No./Substance:
0/Cation Transport Proteins; 0/Codon; 0/DNA-Binding Proteins; 0/ERG protein, human; 0/ERG1 potassium channel; 0/Ether-A-Go-Go Potassium Channels; 0/KCNH6 protein, human; 0/Potassium Channels; 0/Potassium Channels, Voltage-Gated; 0/Trans-Activators

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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