Document Detail


Analysis of the UDP-glucuronosyltransferase gene in Portuguese patients with a clinical diagnosis of Gilbert and Crigler-Najjar syndromes.
MedLine Citation:
PMID:  16269258     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
We describe the molecular study in a cohort of 120 Portuguese patients with the clinical diagnosis of Gilbert syndrome and in one with the diagnosis of Crigler-Najjar syndrome type II, as well as a prenatal diagnosis of Crigler-Najjar syndrome type I. Among the 120 unrelated patients with Gilbert syndrome, 110 were homozygous for the [TA]7 allele ([TA]7/[TA]7), and one patient was a compound heterozygote for two different insertions ([TA]7/[TA]8). The remaining 9 patients were heterozygous for the TA insertion ([TA]6/[TA]7). Additional studies in these 9 patients revealed heterozygosity for the c.674T>G, c.488_491dupACCT and c.923G>A mutations, in 1, 1 and 4 patients, respectively. The patient with Crigler-Najjar syndrome type II was a compound heterozygote for [TA]7 and the c.923G>A mutation. The undocumented polymorphisms c.-1126C>T and c.997-82T>C were also detected in the course of this study. Prenatal diagnosis in a family with a boy previously diagnosed as Crigler-Najjar syndrome type I and homozygosity for the c.923G>A mutation revealed that the fetus was unaffected. Homozygosity for the [TA] insertion was found to be the most frequent cause of GS in our population. Identification of further mutations in the UGT1A1 gene was also seen to contribute significantly towards diagnosis.
Authors:
Elísio Costa; Emília Vieira; Marcia Martins; Jorge Saraiva; Eugénia Cancela; Miguel Costa; Roswitha Bauerle; Teresa Freitas; João R Carvalho; Ermelinda Santos-Silva; José Barbot; Rosário Dos Santos
Related Documents :
15367858 - Congenital myasthenic syndromes.
11843828 - Cholelithiasis and gilbert's syndrome in homozygous beta-thalassaemia.
17162528 - Mortality and pathological findings in c (opitz trigonocephaly) syndrome.
18763178 - A novel splice site mutation in the eya1 gene in a korean family with branchio-oto (bo)...
22508068 - From furuncle to axillary web syndrome: shedding light on histopathology and pathogenesis.
20017898 - Beta2 adrenergic agonists in acute lung injury? the heart of the matter.
Publication Detail:
Type:  Journal Article     Date:  2005-11-02
Journal Detail:
Title:  Blood cells, molecules & diseases     Volume:  36     ISSN:  1079-9796     ISO Abbreviation:  Blood Cells Mol. Dis.     Publication Date:    2006 Jan-Feb
Date Detail:
Created Date:  2006-01-13     Completed Date:  2006-05-04     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9509932     Medline TA:  Blood Cells Mol Dis     Country:  United States    
Other Details:
Languages:  eng     Pagination:  91-7     Citation Subset:  IM    
Affiliation:
Escola Superior de Saúde, Instituto Politécnico de Bragança, Avenida D. Afonso V, 5300-121 Bragança, Portugal. elisio.costa@ipb.pt
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Adolescent
Adult
Child
Crigler-Najjar Syndrome / diagnosis,  enzymology,  genetics*
Female
Gilbert Disease / diagnosis,  enzymology,  genetics*
Glucuronosyltransferase / genetics*,  metabolism
Humans
Male
Middle Aged
Mutagenesis, Insertional
Point Mutation*
Polymorphism, Single Nucleotide*
Portugal
Chemical
Reg. No./Substance:
EC 2.4.1.-/bilirubin uridine-diphosphoglucuronosyl transferase 1A1; EC 2.4.1.17/Glucuronosyltransferase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Association of cardiovascular risk factors with pattern of lower limb atherosclerosis in 2659 patien...
Next Document:  Opsoclonus-myoclonus in an HIV-infected child on antiretroviral therapy--possible immune reconstitut...