Document Detail


Amino-terminal phosphorylation of activation-induced cytidine deaminase suppresses c-myc/IgH translocation.
MedLine Citation:
PMID:  21135131     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Activation-induced cytidine deaminase (AID) is a mutator enzyme that initiates class switch recombination and somatic hypermutation of immunoglobulin genes (Ig) in B lymphocytes. However, AID also produces off-target DNA damage, including mutations in oncogenes and double-stranded breaks that can serve as substrates for oncogenic chromosomal translocations. AID is strictly regulated by a number of mechanisms, including phosphorylation at serine 38 and threonine 140, which increase activity. Here we show that phosphorylation can also suppress AID activity in vivo. Serine 3 is a novel phospho-acceptor which, when mutated to alanine, leads to increased class switching and c-myc/IgH translocations without affecting AID levels or catalytic activity. Conversely, increasing AID phosphorylation specifically on serine 3 by interfering with serine/threonine protein phosphatase 2A (PP2A) leads to decreased class switching. We conclude that AID activity and its oncogenic potential can be downregulated by phosphorylation of serine 3 and that this process is controlled by PP2A.
Authors:
Anna Gazumyan; Ksenia Timachova; Grace Yuen; Edward Siden; Michela Di Virgilio; Eileen M Woo; Brian T Chait; Bernardo Reina San-Martin; Michel C Nussenzweig; Kevin M McBride
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2010-12-06
Journal Detail:
Title:  Molecular and cellular biology     Volume:  31     ISSN:  1098-5549     ISO Abbreviation:  Mol. Cell. Biol.     Publication Date:  2011 Feb 
Date Detail:
Created Date:  2011-01-17     Completed Date:  2011-02-24     Revised Date:  2011-08-03    
Medline Journal Info:
Nlm Unique ID:  8109087     Medline TA:  Mol Cell Biol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  442-9     Citation Subset:  IM    
Affiliation:
Laboratory of Molecular Immunology, Rockefeller University, 1230 York Ave., New York, NY 10065, USA.
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MeSH Terms
Descriptor/Qualifier:
Amino Acid Sequence
Amino Acid Substitution / genetics
Animals
Cytidine Deaminase / chemistry*,  metabolism*
Enzyme Inhibitors / pharmacology
Fibroblasts / drug effects,  enzymology
Immunoglobulin Class Switching / drug effects
Immunoglobulin Heavy Chains / genetics*
Mice
Molecular Sequence Data
Mutant Proteins / metabolism
Phosphorylation / drug effects
Phosphoserine / metabolism
Protein Phosphatase 2 / antagonists & inhibitors,  metabolism
Proto-Oncogene Proteins c-myc / genetics*
Recombination, Genetic / drug effects,  genetics
Somatic Hypermutation, Immunoglobulin / drug effects,  genetics
Translocation, Genetic* / drug effects
Grant Support
ID/Acronym/Agency:
R01AI03752616/AI/NIAID NIH HHS; RR00862/RR/NCRR NIH HHS
Chemical
Reg. No./Substance:
0/Enzyme Inhibitors; 0/Immunoglobulin Heavy Chains; 0/Mutant Proteins; 0/Proto-Oncogene Proteins c-myc; 17885-08-4/Phosphoserine; EC 3.1.3.16/Protein Phosphatase 2; EC 3.5.4.-/AICDA (activation-induced cytidine deaminase); EC 3.5.4.5/Cytidine Deaminase
Comments/Corrections

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