Document Detail


Alterations in hippocampal GAP-43, BDNF, and L1 following sustained cerebral ischemia.
MedLine Citation:
PMID:  12062469     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Alterations in factors involved in the regeneration of the neuronal network in the hippocampus of rats with microsphere embolism (ME) were examined. Nine hundred microspheres (48 microm in diameter) were injected into the right hemisphere, and immunochemical and immunohistochemical studies on the hippocampus were performed on the seventh day thereafter. Hematoxylin-eosin staining showed progressive and severe degeneration of the hippocampus after ME. The protein levels of brain-derived neurotrophic factor (BDNF), 43-kDa growth-associated protein (GAP-43), and adhesion protein L1 (L1) in the ipsilateral hippocampus of the ME animal, determined by Western blot analysis or enzyme immunoassay, were increased, unaltered, and decreased, respectively. In contrast, the immunohistochemical study showed increases in a marker of axonal sprouting GAP-43, and a neurotrophic factor BDNF, and a decrease in an adhesion molecule L1 in some areas of the hippocampal ischemic penumbra of such animals. These results suggest that some factors for regeneration of the neuronal network in the ischemic penumbra responded to sustained cerebral ischemia for a certain period, although functional network of the nerve cells in the microsphere-injected hemisphere would be unlikely established after ME.
Authors:
Keiko Miyake; Wataru Yamamoto; Mina Tadokoro; Norio Takagi; Kyoko Sasakawa; Atsumi Nitta; Shoei Furukawa; Satoshi Takeo
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Brain research     Volume:  935     ISSN:  0006-8993     ISO Abbreviation:  Brain Res.     Publication Date:  2002 May 
Date Detail:
Created Date:  2002-06-13     Completed Date:  2002-08-07     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  0045503     Medline TA:  Brain Res     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  24-31     Citation Subset:  IM    
Affiliation:
Department of Pharmacology, Tokyo University of Pharmacy and Life Science, 1432-1 Horinouchi, 192-0392 Hachioji, Japan.
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MeSH Terms
Descriptor/Qualifier:
Animals
Brain Ischemia / metabolism*,  pathology,  physiopathology
Brain-Derived Neurotrophic Factor / metabolism*
Carotid Artery Thrombosis / metabolism,  pathology,  physiopathology
Cell Death / physiology
Cerebral Infarction / metabolism*,  pathology,  physiopathology
Cerebrovascular Circulation / physiology
Disease Models, Animal
Down-Regulation / physiology
GAP-43 Protein / metabolism*
Hippocampus / metabolism*,  pathology,  physiopathology
Immunohistochemistry
Leukocyte L1 Antigen Complex
Male
Membrane Glycoproteins / metabolism*
Microspheres
Nerve Degeneration / metabolism,  pathology,  physiopathology
Nerve Regeneration / physiology*
Neural Cell Adhesion Molecules / metabolism*
Rats
Rats, Wistar
Time Factors
Up-Regulation / physiology
Chemical
Reg. No./Substance:
0/Brain-Derived Neurotrophic Factor; 0/GAP-43 Protein; 0/Leukocyte L1 Antigen Complex; 0/Membrane Glycoproteins; 0/Neural Cell Adhesion Molecules

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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