Document Detail


All amacrine cells in the rabbit retina possess AMPA-, NMDA-, GABA-, and glycine-activated currents.
MedLine Citation:
PMID:  15259569     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Physiological properties of ligand-activated currents were characterized for morphologically identified AII amacrine cells in the rabbit retina by using whole-cell recordings in a superfused retina slice preparation. The AII amacrine cells were identified based on their distinct narrow-field, bistratified morphology. In the present study, the whole-cell recordings from AII amacrine cells synaptically isolated from presynaptic influences demonstrated the presence of glutamate AMPA (alpha-amino-3-hydroxy-5-methyl-4-isoxazole-propionic acid) receptors, but no kainate receptors. The presence of only AMPA receptors on rabbit AII amacrine cells is in contrast to an earlier study on rabbit AII amacrine cells by Bloomfield and Xin (2000), but consistent with previous studies on rat AII amacrine cells. In addition, NMDA (N-methyl-D-aspartate) -activated currents blocked by the NMDA antagonist D-AP7 (D-2-amino-7-phosphonoheptanoic acid) were found on the AII amacrine cells. These most likely extrasynaptic NMDA-activated currents were attenuated by the presence of Co2+ interacting with Mg2+ and Ca2+ as they competed for divalent cation-binding sites within the NMDA channel. AII amacrine cells also possessed GABA (gamma-aminobutyric acid) -activated currents that were unaffected by the GABAc receptor antagonist TPMPA (1,2,5,6-tetrahydropyridine-4-yl methylphosphinic), but were completely blocked by the GABA(A) antagonist bicuculline. This indicates that the major inhibitory inputs were mediated by only GABA(A) receptors located directly on the AII amacrine cells. Furthermore, although the AII amacrine cells were glycinergic amacrine cells, they also possessed glycine-activated currents that may be mediated by autoreceptors.
Authors:
Chengwen Zhou; Ramon F Dacheux
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Visual neuroscience     Volume:  21     ISSN:  0952-5238     ISO Abbreviation:  Vis. Neurosci.     Publication Date:    2004 Mar-Apr
Date Detail:
Created Date:  2004-07-19     Completed Date:  2004-08-12     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  8809466     Medline TA:  Vis Neurosci     Country:  England    
Other Details:
Languages:  eng     Pagination:  181-8     Citation Subset:  IM    
Affiliation:
Department of Ophthalmology, University of Alabama at Birmingham, Birmingham 35294-0009, USA.
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MeSH Terms
Descriptor/Qualifier:
Amacrine Cells / drug effects,  metabolism*
Animals
Cell Membrane / drug effects,  metabolism
Electrophysiology
Excitatory Amino Acid Agonists / pharmacology
Excitatory Amino Acid Antagonists / pharmacology
Patch-Clamp Techniques
Rabbits
Receptors, AMPA / metabolism*
Receptors, GABA / metabolism*
Receptors, Glycine / metabolism*
Receptors, N-Methyl-D-Aspartate / metabolism*
Retina / metabolism*
Grant Support
ID/Acronym/Agency:
EY-03039/EY/NEI NIH HHS; EY-07033/EY/NEI NIH HHS; EY03011/EY/NEI NIH HHS
Chemical
Reg. No./Substance:
0/Excitatory Amino Acid Agonists; 0/Excitatory Amino Acid Antagonists; 0/Receptors, AMPA; 0/Receptors, GABA; 0/Receptors, Glycine; 0/Receptors, N-Methyl-D-Aspartate

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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