Document Detail


Alcoholic neuropathy is clinicopathologically distinct from thiamine-deficiency neuropathy.
MedLine Citation:
PMID:  12838517     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Characteristics of alcoholic neuropathy have been obscured by difficulty in isolating them from features of thiamine-deficiency neuropathy. We assessed 64 patients with alcoholic neuropathy including subgroups without (ALN) and with (ALN-TD) coexisting thiamine deficiency. Thirty-two patients with nonalcoholic thiamine-deficiency neuropathy (TDN) also were investigated for comparison. In ALN, clinical symptoms were sensory-dominant and slowly progressive, predominantly impairing superficial sensation (especially nociception) with pain or painful burning sensation. In TDN, most cases manifested a motor-dominant and acutely progressive pattern, with impairment of both superficial and deep sensation. Small-fiber-predominant axonal loss in sural nerve specimens was characteristic of ALN, especially with a short history of neuropathy; long history was associated with regenerating small fibers. Large-fiber-predominant axonal loss predominated in TDN. Subperineurial edema was more prominent in TDN, whereas segmental de/remyelination resulting from widening of consecutive nodes of Ranvier was more frequent in ALN. Myelin irregularity was greater in ALN. ALN-TD showed a variable mixture of these features in ALN and TDN. We concluded that pure-form of alcoholic neuropathy (ALN) was distinct from pure-form of thiamine-deficiency neuropathy (TDN), supporting the view that alcoholic neuropathy can be caused by direct toxic effect of ethanol or its metabolites. However, features of alcoholic neuropathy is influenced by concomitant thiamine-deficiency state, having so far caused the obscure clinicopathological entity of alcoholic neuropathy.
Authors:
Haruki Koike; Masahiro Iijima; Makoto Sugiura; Keiko Mori; Naoki Hattori; Hiroki Ito; Masaaki Hirayama; Gen Sobue
Related Documents :
7131047 - Differentiation of the nodal and internodal axolemma in the optic nerves of neonatal rats.
2083877 - Study of innervation of the pancreatic duct of lepus europaeus, using the cholinesteras...
23059557 - Myogenin, myod and igf-i regulate muscle mass but not fiber-type conversion during resi...
55467 - Dynamic aspects of peripheral nerve changes in progressive neural muscular atrophy: lig...
21888527 - Forearm muscle size, strength, force, and power in relation to pqct-derived bone streng...
2403277 - Maximal isometric torque of knee extension as a function of muscle length in subjects o...
Publication Detail:
Type:  Comparative Study; Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Annals of neurology     Volume:  54     ISSN:  0364-5134     ISO Abbreviation:  Ann. Neurol.     Publication Date:  2003 Jul 
Date Detail:
Created Date:  2003-07-02     Completed Date:  2003-08-20     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  7707449     Medline TA:  Ann Neurol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  19-29     Citation Subset:  IM    
Affiliation:
Department of Neurology, Nagoya University, Graduate School of Medicine, Nagoya, Japan.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Adult
Aged
Aged, 80 and over
Alcoholic Neuropathy / chemically induced,  complications,  diagnosis*
Axons / pathology
Biopsy
Ethanol / adverse effects
Female
Humans
Male
Median Nerve / pathology,  physiopathology
Middle Aged
Muscle, Skeletal / innervation,  physiopathology
Myelin Sheath / pathology
Nerve Fibers / pathology
Nociceptors / physiology
Pain / complications
Ranvier's Nodes / pathology
Sural Nerve / pathology,  physiopathology
Thiamine Deficiency / complications,  diagnosis*,  physiopathology*
Tibial Nerve / pathology,  physiopathology
Chemical
Reg. No./Substance:
64-17-5/Ethanol

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Hereditary motor and sensory neuropathy with agenesis of the corpus callosum.
Next Document:  Nonsyndromic mental retardation and cryptogenic epilepsy in women with doublecortin gene mutations.