| Albright's hereditary osteodystrophy with extensive heterotopic ossification of the oral and maxillofacial region: how fetuin research may help a seemingly impossible condition. | |
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MedLine Citation:
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PMID: 18028761 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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Albright"s hereditary osteodystrophy (AHO) is a complex genetic disorder characterized by brachydactyly, gonadotropin resistance, hypothyroidism, pseudohypoparathyroid syndrome and heterotopic ossification. Heterotopic ossification rarely occurs in the maxillofacial region. In this article, we present such a case, describe the etiology, characteristics and treatment of AHO and suggest a potential role of an inhibitor of bone formation such as fetuin in preventing recurrence of aberrant ossification. |
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Authors:
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Marc G DuVal; Sarah Davidson; Andrew Ho; Rachale Cohen; Michael Park; Somayeh Nourian; Gerald Baker; George K B Sándor |
Publication Detail:
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Type: Case Reports; Journal Article |
Journal Detail:
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Title: Journal (Canadian Dental Association) Volume: 73 ISSN: 1488-2159 ISO Abbreviation: J Can Dent Assoc Publication Date: 2007 Nov |
Date Detail:
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Created Date: 2007-11-21 Completed Date: 2007-12-12 Revised Date: - |
Medline Journal Info:
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Nlm Unique ID: 7907605 Medline TA: J Can Dent Assoc Country: Canada |
Other Details:
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Languages: eng Pagination: 845-50 Citation Subset: D; IM |
Affiliation:
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Mount Sinai Hospital, Toronto, Ontario, Canada. |
Export Citation:
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APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
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Adolescent Ankylosis / etiology Bone Morphogenetic Proteins / metabolism Fibrous Dysplasia, Polyostotic / complications* Humans Male Masticatory Muscles / physiopathology Maxillofacial Abnormalities / etiology* Ossification, Heterotopic / etiology*, prevention & control Osteogenesis / physiology Pseudopseudohypoparathyroidism / complications* Temporomandibular Joint Disorders / etiology Transforming Growth Factor beta1 / metabolism alpha-Fetoproteins / physiology* |
| Chemical | |
Reg. No./Substance:
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0/Bone Morphogenetic Proteins; 0/Transforming Growth Factor beta1; 0/alpha-Fetoproteins |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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