Document Detail


Aging of different avian cultured cells: lack of ROS-induced damage and quality control mechanisms.
MedLine Citation:
PMID:  19948180     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Elevated reactive oxygen species (ROS) levels have been observed in mammals during aging, implying an important role of ROS in the aging process. Most bird species are known to live longer and to contain lower ROS levels than mammals of the same body weight. The influence of ROS on the aging process of birds has been investigated using pigeon embryonic fibroblasts (PEF) and chicken embryonic fibroblasts (CEF). ROS levels in young avian cells were much lower than in human cells. When cultivated till replicative senescence, PEF proliferated about one-third longer compared to CEF. However, both senescent avian cell populations showed no increased ROS levels or accumulation of ROS-induced damage on the mtDNA or protein level. The investigation for quality control (QC) mechanisms revealed that the autophagosomal/lysosomal pathway was not downregulated in old avian cells and stable overexpression of the autophagy protein ATG5 improved mitochondrial fitness, enhanced the resistance against oxidative stress and prolonged the life span of CEF. Oxidative stress-mediated apoptosis induced a dose-dependent cell proliferation in CEF as well as in PEF. Taken together, our data indicate that autophagy and compensatory proliferation act as QC mechanisms, while ROS did not influence the aging process in avian cells.
Authors:
Valentina Strecker; S?ren Mai; Britta Muster; Sascha Beneke; Alexander B?rkle; J?rgen Bereiter-Hahn; Marina Jendrach
Publication Detail:
Type:  Comparative Study; Journal Article; Research Support, Non-U.S. Gov't     Date:  2009-12-03
Journal Detail:
Title:  Mechanisms of ageing and development     Volume:  131     ISSN:  1872-6216     ISO Abbreviation:  Mech. Ageing Dev.     Publication Date:  2010 Jan 
Date Detail:
Created Date:  2010-01-29     Completed Date:  2010-04-02     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0347227     Medline TA:  Mech Ageing Dev     Country:  Ireland    
Other Details:
Languages:  eng     Pagination:  48-59     Citation Subset:  IM    
Affiliation:
Kinematic Cell Research Group, Institute for Cell Biology and Neuroscience, Center of Excellence Frankfurt: Macromolecular Complexes, Goethe University, Max-von-Laue-Str. 9, 60438 Frankfurt/Main, Germany.
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MeSH Terms
Descriptor/Qualifier:
Animals
Autophagy* / genetics
Cell Aging* / genetics
Cell Proliferation*
Cell Survival
Cells, Cultured
Chick Embryo
Columbidae
DNA Damage
DNA, Mitochondrial / metabolism
Fibroblasts / metabolism*,  pathology
Lysosomes / metabolism
Microtubule-Associated Proteins / genetics,  metabolism
Oxidative Stress*
Protein Carbonylation
Reactive Oxygen Species / metabolism*
Transfection
Chemical
Reg. No./Substance:
0/DNA, Mitochondrial; 0/Microtubule-Associated Proteins; 0/Reactive Oxygen Species

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