Document Detail


Aetiology of icteric hepatitis and fulminant hepatic failure in children and the possible predisposition to hepatic failure by sickle cell disease.
MedLine Citation:
PMID:  2157327     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The aetiological agents of acute icteric hepatitis and fulminant hepatic failure were investigated in 47 children less than 12 years of age presenting at our hospital during the period January to December 1987. Hepatitis A virus was the aetiological agent in 72%, hepatitis B virus in 11%, cytomegalovirus in 2% and non A non B in 15%. These results confirm the endemicity of these viruses in the Saudi population and that they can be contracted in early life and could lead to severe hepatitis. Three of the patients who had hepatitis A virus infection developed fulminant hepatic failure and two of them died. Two of the patients who developed fulminant hepatic failure were also suffering from sickle cell disease. This raises the question of a possible predisposition to hepatic failure in sickle cell disease upon infection with hepatitis A virus.
Authors:
M D Yohannan; M Arif; S Ramia
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Publication Detail:
Type:  Case Reports; Journal Article    
Journal Detail:
Title:  Acta paediatrica Scandinavica     Volume:  79     ISSN:  0001-656X     ISO Abbreviation:  Acta Paediatr Scand     Publication Date:  1990 Feb 
Date Detail:
Created Date:  1990-05-09     Completed Date:  1990-05-09     Revised Date:  2009-11-11    
Medline Journal Info:
Nlm Unique ID:  0000211     Medline TA:  Acta Paediatr Scand     Country:  SWEDEN    
Other Details:
Languages:  eng     Pagination:  201-5     Citation Subset:  IM    
Affiliation:
Department of Paediatrics, College of Medicine, King Saud University, Riyadh, Saudi Arabia.
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MeSH Terms
Descriptor/Qualifier:
Acute Disease
Anemia, Sickle Cell / complications*
Child
Child, Preschool
Cytomegalovirus Infections / etiology
Hepatic Encephalopathy / etiology*
Hepatitis A / etiology
Hepatitis B / etiology
Hepatitis C / etiology
Hepatitis, Viral, Human / etiology
Humans
Infant
Jaundice / etiology
Liver Diseases / etiology*
Male

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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